
You're showering wrong! Doctor reveals common washing error that could be life-threatening
According to German-based vascular health expert Dr Max Maddahali, 'excessively hot' showers can trigger a dangerous drop in blood pressure that could cause you to faint, risking serious injury and even death.
The hot environment causes blood vessels to expand in the skin, an instinctive biological reaction to try and cool the body down.
This process can—in some circumstances—trigger feelings of dizziness and even cause you to fall unconscious.
Fainting can be dangerous at any time but doing in the bathroom could be extremely serious given the number of hard surfaces to hit your head on.
As such, experts advise turning the shower down a few degrees if you find yourself feeling dizzy or installing a temperature lock to avoid the temptation entirely.
Those particularly prone to feelings of dizziness in the shower—such as patients with an existing health condition—may want to have a grab bar, anti-slip or even a stool installed to provide quick support.
Patients can also be recommended to get a wearable medical alert device, which can come as a bracelet, and can raise the alarm if a person falls or finds themselves injured.
But Dr Maddahali said fainting wasn't the only health hazard you risked by having a hot shower.
In a TikTok clip which has been viewed over 14million times, he also said it could also pose a risk to your skin, hair and even your immune system.
Dr Maddahali said: 'There's some natural on your skin that are keeping your skin moist.'
'Hot water can wash them off completely so dryness can happen.
'Also, too hot water can cause hair damage.'
Scalding water damages hair much in the same way it damages skin, by stripping it of natural oils that protect making it more vulnerable to damage.
While Dr Maddahali didn't explain how hot showers weaken the immune system—our body's natural defence against dangerous virus and bacteria—some research suggests this is true.
Hot water is more effective at removing a natural layer of 'good' bacteria that live naturally on the skin.
Some experts suggest removing this layer leads to a less active immune system, which could reduce its ability to respond to actual threats.
Social media users reacting to the clip said they would keep their steaming hot showers, despite the medic's advice.
'I respectfully will not take your advice,' one wrote.
Another added: 'It is therapeutic, hot showers keep me sane.'
Medics have provided a range of advice about showers over the years, from when to take them to what to never do in them.
While the nation may be divided about if morning or evening showers reign supreme, experts have ruled a shower before bed to be superior.
Dr Jason Singh, a primary care physician in Virginia, previously highlighted how an evening showers improved sleep quality, washing away pollutants, and hydrating dry skin.
But experts have said you should 'never' urinate in the shower as it could train your brain into associating the act with the sound of running water and leading to incontinence.
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Reuters
a minute ago
- Reuters
UK scientists find genes linked with chronic fatigue syndrome
Aug 6 (Reuters) - Researchers at the University of Edinburgh said on Wednesday they have discovered differences in the DNA of people with chronic fatigue syndrome that should help dispel the notion that the debilitating condition is psychological or driven by laziness. Their study found eight areas of genetic code that are different in people with myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) than in healthy volunteers. The finding provides 'the first robust evidence that genes contribute to a person's chance of developing the disease,' the researchers said in a statement. The key features of the condition include worsening of fatigue, pain and brain fog after even minor physical or mental activity. Very little has been known about the causes of ME/CFS, and there is no diagnostic test or cure. The condition is believed to affect around 67 million people worldwide, the researchers said. The DecodeME study analyzed DNA samples from 15,579 people who reported having chronic fatigue on a questionnaire and 259,909 people without it, all of European descent. Gene variants that were more common in people reporting ME/CFS were linked to the immune and nervous systems, according to a report of the study that has not yet been peer-reviewed. At least two of the gene regions relate to how the body responds to infection, which aligns with reports that the symptoms often start after an infectious illness, the researchers said. Another gene region has previously been identified in people with chronic pain, another common symptom of the condition. The findings 'align with decades of patients reporting on their experiences,' researcher Andy Devereux-Cooke said in a statement, adding that they "should prove game changing in the ME/CFS research field.' 'These results will not mean that a test or cure will be developed straight away, but they will lead to a greater understanding," he said. Scientists who were not involved in the study said using volunteers who self-reported chronic fatigue syndrome rather than restricting participation to those with a diagnosis from a medical professional somewhat weakened its conclusions. They called for larger studies to replicate the results. Substantial work will be necessary 'to translate these findings into new treatments," said Dr. Jackie Cliff, who studies infection and immunity in ME/CFS at Brunel University of London. "This will take considerable investment in academia and by industry.'


The Guardian
an hour ago
- The Guardian
Scientists find link between genes and ME/chronic fatigue syndrome
Scientists have found the first robust evidence that people's genes affect their chances of developing myalgic encephalomyelitis or chronic fatigue syndrome (ME/CFS), a mysterious and debilitating illness that has been neglected and dismissed for decades by many in the medical community. Early findings from the world's largest study into the genetics of the condition pinpointed eight regions of the human genome that were substantially different in people with an ME/CFS diagnosis compared to those without the illness. The discovery suggests that several variants of genes commonly found in the population raise the risk of developing the illness, though many people will carry the variants and never acquire it. Prof Chris Ponting, an investigator on the DecodeME study at the University of Edinburgh, called the results 'a wake-up call' that showed a person's genetics could 'tip the balance' on whether they would develop ME/CFS. 'These provide the first robust evidence for genetic contributions to ME,' Ponting said. 'There are many genetic variants that apply across the genome that predispose people to be diagnosed with ME.' More research is needed to develop diagnostic tests or screenings to identify people at high risk of ME/CFS. But scientists called the work a milestone that put the illness on an equal footing with other debilitating diseases and opened potential avenues for treatments. 'This really adds validity and credibility for people with ME,' said Sonya Chowdhury, chief executive of Action for ME and a DecodeME co-investigator. 'We know that many people have experienced comments like 'ME is not real'. They've been to doctors and they've been disbelieved or told that it's not a real illness.' Despite its long history, scientists understand very little about the causes of ME/CFS, though most patients report an infection before symptoms first appear. Typical symptoms include extreme tiredness, sleep problems, brain fog and a worsening of symptoms after physical or mental activities, known as post-exertional malaise, which can take weeks to recover from. It is estimated that 67 million people are affected by ME/CFS at an annual cost to the global economy of tens of billions of pounds. In the UK, the annual economic toll is calculated at more than £3bn. There is no test or cure for the illness. The DecodeME study, a collaboration between Edinburgh University, ME charities and patients, was launched in 2022 to explore whether genes play a role in who develops ME/CFS. For the latest work, researchers analysed 15,579 DNA samples from 27,000 people with ME/CFS and more than 250,000 people without the illness. The eight genetic regions that stood out in people with ME/CFS contain genes involved in immune defences and the nervous system. It will take more work to unpick the biology, but some gene variants may make people more vulnerable to ME/CFS by compromising their ability to fight bacterial and viral infections. Another genetic difference seen in ME/CFS is known from people with chronic pain, a symptom that many with ME/CFS also experience. 'Overall, what is happening here is the genetics align with how people with ME have described their illness,' Ponting said. Andy Devereux-Cooke, a DecodeMe co-investigator, said the findings would be huge for patients. 'The vast majority of the patient population essentially has been abandoned in one way or another, by families, the government, the medical system,' he said. 'This will be huge for the patient population. Even though it does not provide all the answers [and] it does not provide practical assistance, it is a welcome drop in the ocean towards turning the tide.' Among the many questions that remain is why ME/CFS affects far more women than men. Diagnoses are four times more common in women, but the study found no genetic explanation. Another question is whether long Covid overlaps with ME/CFS. While many symptoms are similar, the researchers found no genetic link between the two. 'One of the key things we're doing is enabling others to use their different approaches to ask and answer the same question,' said Ponting. Prof Anne McArdle, who studies ME/CFS at the University of Liverpool, said the results, which have not yet been published in a peer-reviewed journal, provided 'a solid basis' for future work that would hopefully help accelerate the development of a treatment for the devastating illness. Dr Beata Godlewska, who studies ME/CFS at the University of Oxford, recently used magnetic resonance spectroscopy to scan the brains of people with ME/CFS and long Covid. Those with ME/CFS but not long Covid had high levels of lactate in the anterior cingulate cortex, a brain region that integrates information about effort and emotion. This points to disrupted energy metabolism in the brain, and impaired mitochondria, the battery-like structures that provide energy inside cells. Godlewska said 'It's a very sad fact that people with ME/CFS are still disbelieved and the disease has been so neglected, especially when it comes to research funding. Hopefully this study will come with a benefit of both fighting the stigma, and convincing research funders that this is a truly biological condition.'


The Guardian
an hour ago
- The Guardian
A moment that changed me: on the day of my first book deal, a mysterious hum overcame me
I developed tinnitus the same night I was offered my first book deal at the end of 2014. I'd received the news late-afternoon, then went out for cocktails with two friends to celebrate. I remember the evening well: we'd gone somewhere loud but not too loud; I ordered a fluorescent orange drink that I didn't enjoy. Before I went to bed, I spoke to my boyfriend on the phone. 'This could really change your life,' he said. Sometime in the early hours of the following morning, I woke up with a ringing in my ears that has not gone away since. Tinnitus often appears out of nowhere. Some describe it as like standing next to the engine of a plane, or caught within a colony of flying bats. The sound I woke with wasn't so bad: a sort of midpoint between the hiss of television static and the roar of the ocean. At first I thought it must have been coming from my phone, which was usually inches away from my face as I slept. I then searched my room for malfunctioning electronics, before realising with bracing horror that the noise was coming from inside my brain. The condition can be divided into three categories: subjective tinnitus (the perception of sound where there is none); objective tinnitus (where there is a sound within or close to the ear that another person can sometimes hear); and somatic tinnitus, which worsens with body movement. Mine was the former: a phantom sound. Little is known about what causes it, but one theory is that loud sounds damage the hairs in the cochlea, flattening them like stems of corn after a storm. The hairs normally pick up sounds and, without them, the brain seeks out sounds from other parts of the ear, sending back signals that translate to a perpetual illusory hum. I'd spent many years going to loud concerts and playing drums in a band, only occasionally protecting my hearing with earplugs. I started to blame myself. The first few days felt long and hard. It seemed absurd to be so troubled by what is basically an annoying sound, but its inescapability and totality was maddening. The world was suddenly bifurcated into quiet spaces, in which I had nothing to mask the tinnitus, and loud spaces, which threatened to damage my hearing further. I couldn't stop catastrophising: 'I will never hear silence again. I will always be too distracted to write. I will be driven mad by loud sounds.' I became anxious and, like 21% of tinnitus patients, I developed suicidal thoughts. My GP was unsympathetic, telling me it would not go away but I would get used to it – and no, I couldn't have any Valium. A few days before Christmas, I went to stay with my boyfriend's parents for the first time. Ironically, there were copies of the Max Ehrmann poem Desiderata all over the house – printed off in the guest room, hanging beside the bathroom sink. It was one of his mum's favourites; she identified strongly with the message: 'Go placidly amid the noise … remember what peace there may be in silence.' I read online studies about tinnitus compulsively. I discovered advice to seek out quiet spaces in which I could place my hands over my ears to get a measure of whether it was getting any better or worse. But this only exacerbated my nerves. On New Year's Day, I couldn't stop crying; I felt stuck in an abject state of misery and panic. I began hypnotherapy, hoping I could trick my brain into not hearing the never-ending noise. My hypnotherapist told me this was not how it worked. Instead, she tried to teach me how to separate my feelings of anxiety from the sounds in my ears, and how to relax; sending me away with audio files of women whispering about the sea. I began sleeping to the sound of brown noise – named after 'Brownian motion', the random movement of particles in a liquid – replacing one hum with another. I practised progressive muscle relaxation, my antidepressants kicked in and I began the long process of habituation – learning to tune out the unwanted, unstoppable sound. Just over a month after I first developed tinnitus, I was on holiday in Brasov, Transylvania – a 30th-birthday present from my sister and mum. Towards the end of the trip, the three of us were walking through the mountains, and when I looked back I realised I couldn't see either of them. I was surrounded only by recently settled snow. There was something about the white of the snow and the ringing in my ears that felt equivalent; blank and unintimidating, somehow. Tinnitus is often described as white noise, but white is achromatic – a colour that isn't really a colour. I knew then that I'd make peace with this sound that isn't really a sound, my new version of silence. That was a decade ago, and these days I've learned to accept and adjust. I'd railed so violently against my tinnitus, and achieved nothing. Now I acknowledge what I cannot change, and it doesn't bother me at all. In the UK and Ireland, Samaritans can be contacted on freephone 116 123, or email jo@ or jo@ In the US, you can call or text the National Suicide Prevention Lifeline on 988, chat on or text HOME to 741741 to connect with a crisis counselor. In Australia, the crisis support service Lifeline is 13 11 14. Other international helplines can be found at Do you have an opinion on the issues raised in this article? If you would like to submit a response of up to 300 words by email to be considered for publication in our letters section, please click here. This article was amended on 6 August 2025 to correct the quotation from Max Ehrmann's poem Desiderata.