
Yani Tseng hopes to rediscover ‘passion' at US Open after overcoming the yips
Five-time major winner Yani Tseng will compete in her first US Open since 2016 this week after overcoming the dreaded 'yips' by putting left-handed.
Tseng burst onto the scene in 2008 by winning the Women's PGA Championship and collected all five of her majors in a four-year spell which saw her reach the top of the world rankings in 2011 and 2012.
Three wins in the space of five weeks at the start of 2012 were her last to date on the LPGA however – she did win in her native Taiwan in 2014 – and Tseng's slow decline saw her slump outside the world's top 100 in 2017.
Tseng stepped away from the sport for almost two full years in 2019 due to a combination of a back injury and the Covid-19 pandemic, and when she worked hard to return felt that her putting woes meant she effectively needed to hole her approach shots to make up any ground on the competition.
After missing the cut in all nine LPGA Tour starts in 2021 Tseng did not compete on the circuit in 2022 or 2023, but returned in April's Chevron Championship, where her switch from putting right-handed to left-handed became apparent.
'I was struggling with my short putts right-handed,' Tseng told the USGA website during a practice round at Erin Hills ahead of the 80th US Women's Open.
'I had the yips.'
Tseng began putting left-handed around six months ago and, using that technique, shot 70-71 in a 36-hole US Open qualifier at Arizona Country Club before claiming the only place in this week's field from a five-woman play-off.
'The passion never went away,' the 36-year-old added. 'The past few years I've been disappointed with my performance, but I love golf, I love competition, I love the people.
'I want to prove to myself that I can still be a player at this level. I want to see how far I can go.'
As a past champion of the KPMG Women's PGA Championship and the AIG Women's Open, Tseng is eligible for those majors and plans to play them this year, at Fields Ranch East in Texas and Royal Porthcawl respectively.
She has been working with coach Kristine Reese from the Vision 54 programme run by Lynn Marriott and Pia Nilsson, who can count Annika Sorenstam and former Solheim Cup captain Suzann Pettersen among their pupils.
'What I need to focus is inside myself,' Tseng said. 'I need to focus on what I can control, like holding my finish.
'I need to believe in myself. Doubt is the most scary thing. The mechanical and the mental feed off each other.
'Just be yourself, be who you are. Keep looking into yourself and seeing the good things. I tried to be perfect all the time. That's not a way to live.'

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The Guardian
an hour ago
- The Guardian
For the USMNT, a successful summer is harder than ever to define
In a certain sense, there really is no winning the Concacaf Gold Cup. Not if you're the United States men's national team, at any rate. While the tournament's name may allude to a glory conferred by the most valuable of precious metals, the whole thing remains among the ugly ducklings of global continental championships. If you're the US, lifting the biennial Gold Cup this summer would amount to winning it for an eighth time overall and a sixth in a quarter century. There's no novelty to it, no real sense of upward momentum on the long-sought ascent to a higher international plane. Fail to win it, however, and there will be an inquest and existential questions, even though this incarnation of the American roster is missing a half dozen-or-so key pieces, depending on how you count them. But if the optics of the Gold Cup are zero-sum, it retains an intrinsic value to the Yanks less in the thing itself than in what it simulates: a World Cup. Squint, and pretend that you're playing, say, Poland and South Korea instead of Haiti and Trinidad and Tobago, and there's something useful in going through the cadence of a competitive summer tournament. Even if the opponents are just the same old regional foes – fellow Group D denizens Saudi Arabia excepted, as they're a guest team – the slow boil and gathering momentum of an unspooling tournament offers helpful experience. Certainly, the hope was that the 2025 Gold Cup would be an exercise in putting the finishing touches on a supposed golden generation, positioning all the pieces just so as the team prepared for a career-defining summer next year while hosting the 2026 World Cup. Going through a tournament one last time would be a final chance to spot and address the flaws and fissures in the foundation. Winning it, if at all possible, would be a kind of byproduct of all that finessing and finetuning. Instead, the Americans will spend the summer trying to rehabilitate their battered reputation after March's Concacaf Nations League debacle. The discourse will probably be dominated by who isn't there as much as who is. Yet there can still be some real use in this exercise. It's even feasible that the USMNT could have a good summer by showing some signs of life. 'I don't think there's any denying that some of our performances have fallen short over the past year to 18 months,' said defender Walker Zimmerman. 'It's something that us, as players, we obviously aren't satisfied with, and it's a big focal point for this camp. It's always such a great opportunity to have a month in front of the staff, get a lot of quality trainings in together, and find yourself hopefully getting into a rhythm of playing multiple games where you can put everything on the line to try to make a World Cup team in a year's time. It's a massive opportunity.' All the absences underscore just how prone a summer tournament is to the flukes of form and fitness. But head coach Mauricio Pochettino has a chance to scour his overhauled roster for a few new depth pieces and tactical alternatives in among all the fresh faces, sourced in unexpected number from Major League Soccer. The USMNT's lineup has been largely ossified going back a half decade or so now, and when given a chance, more pleasant surprises like Real Salt Lake's spitfire midfielder Diego Luna might well present themselves. Or players who can offer the American attack a different look anyway, on the semi-regular days when Plan A isn't working. Sign up to Soccer with Jonathan Wilson Jonathan Wilson brings expert analysis on the biggest stories from European soccer after newsletter promotion Then there are the players who have accomplished resumes at the club level but, for whatever reason, just not really shown it for the national team yet. Malik Tillman, the Bayern Munich castoff, has established himself as one of the best players in the Dutch Eredivisie in an attacking midfielder role where the U.S. could really use a spark. While contributing hugely to PSV's second straight league title, however, Tillman has been largely anonymous for the national team. This is also true of the New Jersey-born, Brazilian-raised Johnny Cardoso who, like Tillman, is 23. So impressive in his season-and-a-half with Real Betis has Cardoso been that a move to mighty Atlético Madrid is reportedly imminent. With a full national team complement in camp, Tillman and Cardoso would have gotten lost in the shuffle, either the odd men out in the midfield logjam or pushed out of their best positions. Here, now, is a chance for them to make their case, or at least pose some questions about whether the incumbents really ought to be automatic starters. The importance of identity and tactical systems probably gets overstated at the national team level, where a dearth of time means that an awful lot of teams play broadly the same way. But the coming month does offer Pochettino a chance to cultivate some of the closeness and feistiness that his best club teams displayed. There is perhaps a larger exploration to be undertaken at some point about the ways in which the improved conditions and job security of the modern American player have undercut the existential belligerence the Yanks used to play with, but, for now, any kind of low-level aggravation will do. Some bite. A little more getting-after-it intensity, even when the stadium is half-empty and the opponent unthreatening, on paper at least. There would be as much value in unlocking any of those intangibles as there would in winning the Gold Cup, even if that's the macro expectation. 'I think I'd be lying if I didn't say, 'Lifting that trophy on the final day' would be what we would consider success. I think that's the standard we've set for ourselves,' said goalkeeper Matt Turner. 'But at the same time, things happen in soccer, and I think what we need to control is what we bring to the table every single day: the intensity, the way we push each other, the passion, the energy, the connection with the fans, with each other, with the staff. We're going to be together for a long period of time and it's a really good opportunity for us to put a lot of things together, tactically, technically, emotionally.' Taken together, these alternative accomplishments would probably make for a satisfying summer of national team soccer. They might even compensate for a failure to win the Gold Cup. Leander Schaerlaeckens is at work on a book about the United States men's national soccer team, out in 2026. He teaches at Marist University.


BBC News
an hour ago
- BBC News
The mystery rise of lung cancer in non-smokers
The number of lung cancer cases in people who have never smoked is increasing. The disease is different from lung cancer caused by smoking, so what causes it? Martha first realised that something was wrong when her cough changed and the mucus in her airways became increasingly viscous. Her doctors put it down to a rare disorder she had that caused her lungs to become chronically inflamed. "No worry, it must be that," she was told. When she finally had an X-ray, a shadow was detected on her lung. "That set the ball rolling," Martha recalls. "First, a CT scan was done, then a bronchoscopy [a procedure that involves using a long tube to inspect the airways in a person's lungs] to take tissue samples." After the tumour was removed, about four months after she'd first reported symptoms to her GP, she received the diagnosis: Stage IIIA lung cancer. The tumour had infiltrated the surrounding lymph nodes but had not yet spread to distant organs. Martha was 59 years old. "It was a total shock," says Martha. Although she would occasionally light up a cigarette at a party, she never considered herself a smoker. Lung cancer is the most common cancer worldwide and the leading cause of cancer death. In 2022, about 2.5 million people were diagnosed with the disease and more than 1.8 million died. Although tobacco-related lung cancers still account for the majority of diagnoses worldwide, smoking rates have been declining for several decades. As the number of smokers continues to fall in many countries around the world, the proportion of lung cancer occurring in people who have never smoked is on the rise. Between 10 and 20% of lung cancer diagnoses are now made in individuals who have never smoked. "Lung cancer in never-smokers is emerging as a separate disease entity with distinct molecular characteristics that directly impact treatment decisions and outcomes," says Andreas Wicki, an oncologist at the University Hospital Zurich, Switzerland. While the average age at diagnosis is similar to that of smoking-related lung cancers, younger patients with lung cancer are more likely to have never smoked. "When we see 30- or 35-year-olds with lung cancer, they are usually never-smokers," he says. Another difference is the type of cancer being diagnosed. Until the 1950s and 1960s, the most common form of lung cancer was squamous cell carcinoma – a type which begins with the cells that line the lungs. In contrast, lung cancer in never-smokers is almost exclusively adenocarcinoma – a type which starts in mucus-producing cells – which is now the most common form of lung cancer in both smokers and never-smokers. Like other forms of lung cancer, adenocarcinoma is usually diagnosed at an advanced stage. "If there's a 1cm (0.4in) tumour hidden somewhere in your lungs, you won't notice it," says Wicki. The early symptoms, which include persistent coughing, chest pain, shortness of breath or wheezing, often only appear when the tumour is larger or has spread. In addition, the historically strong link between smoking and lung cancer may inadvertently lead non-smokers to attribute symptoms to other causes, says Wicki. "Most cases in never-smokers are therefore only diagnosed at stage 3 or 4." Lung cancer in never-smokers is also more common in women. Women who have never smoked are more than twice as likely to develop lung cancer as male never-smokers. Aside from lung anatomy and environmental exposures, at least part of the answer may lie in genetic mutations that are more common in women, especially in Asian women. One of the most prevalent is a mutation known as EGFR. Lung cancer cells in people who have never smoked usually have a number of mutations that could be causing their cancer, explains Wicki – so-called driver mutations. These genetic changes drive tumour growth, such as the EGFR gene which codes for a protein on the surface of cells and is called epidermal growth factor receptor. The reasons why these driver mutations are more frequently found in female patients, particularly those of Asian descent, are not entirely understood. There is some evidence that female hormones may play a role, with certain genetic variants that affect oestrogen metabolism being more prevalent in East Asians. This could potentially explain the higher incidence of EGFR-mutant lung cancer in Asian women, although the data is very preliminary. Following the discovery of mutations which can lead to lung cancer in non-smokers, the pharmaceutical industry began to develop drugs that specifically block the activity of those proteins. For example, the first EGFR inhibitors became available around 20 years ago and most patients showed an impressive response. However, treatment often led to resistant cancer cells, resulting in tumour relapse. In recent years, much effort has been put into overcoming this problem, with newer types of drugs now entering the market. As a result, the prognosis for patients has steadily improved. "The median survival rate of patients who carry such driver mutations is now several years," Wicki explains. "We have patients who have been on targeted therapy for more than 10 years. This is a huge step forward when you consider that the median survival rate was less than 12 months about 20 years ago." As the proportion of lung cancer in never-smokers increases, experts say it is crucial to develop prevention strategies for this population. A number of risk factors have been implicated. For example, studies have revealed that radon and second-hand smoke can elevate the risk of lung cancer in non-smokers. Additionally, exposure to cooking fumes or to stoves burning wood or coal in poorly ventilated rooms may also increase this risk. Since women traditionally spend more time indoors, they are particularly vulnerable to this type of indoor air pollution. However, outdoor air pollution is an even more significant factor in the development of lung cancer. In fact, outdoor air pollution is the second leading cause of all lung cancer cases after smoking. Studies have revealed that people who live in highly polluted areas are more likely to die of lung cancer than those who do not. Particulate matter less than 2.5 microns in diameter (about a 30th of the width of a human hair), typically found in vehicle exhaust and fossil fuel smoke, seems to play an important role. And intriguingly, research has shown a strong link between high levels of PM2.5 and lung cancer in individuals who have never smoked and who carry an EGFR mutation. How air pollution may trigger lung cancer in never-smokers carrying the EGFR mutation has been the focus of research at the Francis Crick Institute in London. "When we think about environmental carcinogens, we usually think about them as causing mutations in the DNA", says William Hill, a post-doctoral researcher in the cancer evolution and genome instability laboratory of the Francis Crick Institute. Cigarette smoke, for example, damages our DNA, thus leading to lung cancer. "However, our [2023] study proposes that PM2.5 doesn't directly mutate DNA, rather it wakes up dormant mutant cells sitting in our lungs and starts them on the early stages of lung cancer." In their experiments, the researchers showed that air pollutants are taken up by immune cells called macrophages. These cells normally protect the lung by ingesting infectious organisms. In response to PM2.5 exposure, macrophages release chemical messengers known as cytokines, which wake up cells carrying the EGFR mutation and causes them to proliferate. "Both air pollution and EGFR mutations are needed for tumours to grow," says Hill. Understanding how PM2.5 acts on the microenvironment of cells carrying EGFR mutations to promote tumour growth, he adds, could pave the way for new approaches to preventing lung cancer. The association between air pollution and lung cancer is not new. In a landmark paper establishing the link between smoking and lung cancer in 1950, the authors suggested outdoor pollutants from the burning of fossil fuels as a possible cause. But policies to date have focused almost exclusively on tobacco control. But 75 years later, air pollution is finally coming into focus. Air pollution levels in Europe and the US have fallen in recent decades. But the effect of changes on lung cancer rates has not yet become apparent. "It probably takes 15 to 20 years for changes in exposure to be reflected in lung cancer rates, but we don't know for sure," says Christine Berg, a retired oncologist from the National Cancer Institute in Maryland, US. Moreover, the picture is not static: climate change is likely to have an impact in the future. "With the increasing risk of wildfires, air pollution and PM2.5 levels are rising again in certain regions of the US," says Berg. "At least one study has shown an association between wildfire exposure and increased incidence of lung cancer. Transitioning away from coal, oil and gas is therefore crucial not only to slow global warming but also to improve air quality." In 2021, the WHO halved the annual mean air quality guideline for PM2.5, meaning it has adopted a more stringent approach to particulate matter. "But 99% of the world population lives in areas where air pollution levels exceed [these updated] WHO guideline limits," says Ganfeng Luo, a postdoctoral researcher at the International Agency for Research on Cancer (IARC) in Lyon, France. In a recent study, IARC researchers estimated that approximately 194,000 cases of lung adenocarcinoma worldwide were attributable to PM2.5 in 2022. "The highest burden is estimated in East Asia, especially in China," says Luo. In the future, the number of lung cancer deaths attributable to air pollution could increase in countries such as India, which currently has some of the highest levels of air pollution, according to the WHO. In Delhi, the average PM2.5 levels are above 100 micrograms per sq m, which is 20 times above the WHO air quality guidelines. In the UK, 1,100 people developed adenocarcinoma of the lung as a result of air pollution in 2022, the IARC study found. "But not all of these cases will be in never-smokers," says Harriet Rumgay, an epidemiologist and a co-author of the study. Adenocarcinoma also occurs in smokers, especially in those using filtered cigarettes. "There's still a lot we don't know," says Rumgay. "More research is needed to disentangle the different factors and also to understand, for example, how long you would need to be exposed to air pollution before developing lung cancer." As treatments continue to improve, lung cancer in never-smokers is becoming more survivable. It is conceivable that this type of lung cancer will one day become the most common form of a disease that has historically been associated with older male smokers, changing the way we think about the disease in popular culture; "…the idea that they [patients] are at least partly to blame for their disease is unfortunately still widespread," says Wicki. Martha was found to have an EGFR mutation and has been taking an inhibitor since her diagnosis almost three years ago. "It's definitely not a vitamin pill," she says. The drug has some nasty side effects: chronic fatigue, muscle pain, skin problems. Balancing the risks and benefits of drug treatment and maintaining a reasonable quality of life is not always easy, she says. But the drug is working. "And the fatalistic view of the disease is changing, and that is good." -- For trusted insights into better health and wellbeing rooted in science, sign up to the Health Fix newsletter, while The Essential List delivers a handpicked selection of features and insights. For more science, technology, environment and health stories from the BBC, follow us on Facebook, X and Instagram.


The Sun
an hour ago
- The Sun
UFC 316 – Merab Dvalishvili vs Sean O'Malley 2: Fight time, full card, TV channel and live stream ahead of huge rematch
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