Roseman University Launches $10 Million Medical School Scholarship Campaign
The "Rhythm of the Future" campaign addresses Nevada's critical physician shortage while reducing financial barriers for medical students. Nevada currently ranks 48th nationally in physician-to-patient ratio and faces a shortage of 2,500 physicians compared to the national average.
"This is more than a campaign. It is a movement of generosity, of vision, and of shared responsibility," said Bryan Lindsey, campaign chair and Roseman Board of Trustees member. "The future of healthcare in Nevada depends on what we build together, and this scholarship fund will ensure that talent—not financial status—determines who gets to become a doctor."
The campaign has already secured over $3 million in early commitments, including a $2 million gift from the Engelstad Family Foundation and $500,000 from an anonymous donor.
Dr. Pedro "Joe" Greer Jr., founding dean of Roseman's College of Medicine, emphasized the urgency. "Nevada is 2,500 physicians short of the national average. That's not just a gap. That's a canyon," Greer said. "At Roseman, we are training doctors who will not only treat illness, but who will fight inequity and heal entire systems."
Roseman's College of Medicine distinguishes itself through community-based learning programs including GENESIS and EMPOWERED, which place students in underserved communities from day one.
The campaign coincides with Roseman University's 25th anniversary. Founded in Henderson in 1999 by Drs. Harry Rosenberg, Renee Coffman, and Charles Lacy with $15,000 of personal investment, the university now includes colleges of Pharmacy, Nursing, Dental Medicine, Graduate Studies, and Medicine.
"This scholarship campaign represents the next chapter in our mission—ensuring that every qualified student, regardless of background or circumstance, has the opportunity to become a healer," said Dr. Renee Coffman, president and co-founder.
The College of Medicine, located at Roseman's Summerlin (Las Vegas) campus, received preliminary accreditation from the Liaison Committee on Medical Education in February 2025 and will welcome its charter class of 60 students in July 2025.
Scholarship contributions support tuition, fees, educational materials, and learning experiences. For information, visit roseman.edu/give/give-to-scholarships or contact gmattox@roseman.edu.
Hashtags
Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles
Yahoo
16 hours ago
- Yahoo
Aging Can Spread Through Your Body Via a Single Protein, Study Finds
Take note of the name: ReHMGB1. A new study pinpoints this protein as being able to spread the wear and tear that comes with time as it quietly travels through the bloodstream. This adds significantly to our understanding of aging. Short for reduced high mobility group box 1, ReHMGB1 triggers senescence in cells, permanently disabling them. It doesn't just do this locally; it can send damaging signals throughout the body, particularly in response to injuries or disease. "An important question in aging research is why senescent cells increase with age," write the study authors, led by researchers from the Korea University College of Medicine. Related: The team says their findings could help develop ways to keep us healthier for longer. If we can block or control this protein's signals, it might slow the cascade of cellular decline that comes with age. "This study reveals that aging signals are not confined to individual cells but can be systemically transmitted via the blood, with ReHMGB1 acting as a key driver," says Korea University biomedical engineer Ok Hee Jeon. The researchers were able to identify ReHMGB1 as a critical messenger passing on the senescence signal by analyzing different types of human cells grown in the lab and conducting a variety of tests on mice. When ReHMGB1 transmission was blocked in mice with muscle injuries, muscle regeneration happened more quickly, while the animals showed improved physical performance, fewer signs of cellular aging, and reduced systemic inflammation. The next step would be to see how this process could be disrupted, and this particular type of aging signal kept more localized – so the health conditions that come with old age might not be as damaging. "By blocking this pathway, we were able to restore tissue regenerative capacity, suggesting a promising strategy to treat aging-related diseases," says Jeon. This process is only one contributor to aging out of many, but the signals that ReHMGB1 spreads are particularly important in terms of our bodies becoming dysfunctional over time and less able to carry out repairs. It's also worth bearing in mind the useful functions of ReHMGB1 in the body: alerting our biological systems to damage and indicating that repairs are needed. Any kind of intervention will have to take that into consideration. We know that, generally speaking, populations are living for longer than ever before, and that's putting both our bodies and scientific research into uncharted territory. The various routines built into our cells must continue to function when years ago our bodies would have long given up. Related: From where we live to the genetics we're born with, there are many contributors to aging and lifespan. By learning more about how these work, we'll be better able to manage them – and perhaps extend life further. "Building on current research in these areas will be essential to understanding the therapeutic potential of redox-sensitive HMGB1 in aging-related diseases and its role as a systemic mediator of senescence," write the researchers in their published paper. The research has been published in Metabolism. Related News New Genetic Test Predicts Children With Future Risk of High BMI This Diet Helps Lower Dementia Risk, And We May Finally Know Why A Signal of Future Alzheimer's Could Hide in The Way You Speak Solve the daily Crossword
Yahoo
04-08-2025
- Yahoo
Atos SE (AEXAF) (H1 2025) Earnings Call Highlights: Steady Revenue and Strategic Restructuring ...
Release Date: August 01, 2025 For the complete transcript of the earnings call, please refer to the full earnings call transcript. Positive Points Atos SE (AEXAF) reported a stabilization of revenues at approximately $2 billion per quarter, indicating a steady financial performance. The company achieved a 15% organic growth in operating margin compared to the previous year, showcasing improved operational efficiency. Atos SE (AEXAF) successfully renegotiated a significant contract with a German automotive OEM, turning a previously loss-making contract into a profitable one. The restructuring plan, known as Genesis, is progressing well, with expectations to complete two-thirds of the plan by the end of the year. The company has strengthened its governance with the addition of a new board member, enhancing leadership and oversight. Negative Points Atos SE (AEXAF) reported a net income group share of minus 696 million, indicating significant financial challenges. The company's net debt increased to 1.7 billion, reflecting a higher leverage ratio of 4.0 times. The book-to-bill ratio remains below 100%, raising concerns about future revenue growth and contract renewals. The company incurred reorganization and rationalization charges totaling 379 million, impacting overall profitability. Atos SE (AEXAF) faces ongoing challenges with loss-making contracts, particularly in the UK, which continue to affect cash flow. Q & A Highlights Warning! GuruFocus has detected 5 Warning Signs with AEXAF. Q: Can you discuss the revenue trajectory for the rest of the year and any specific assumptions regarding demand and seasonality? A: Unidentified_1: We expect Q3 revenues to stabilize around $2 billion, slightly less than previous quarters, with an increase anticipated in Q4. The revenue stabilization is crucial, and we have successfully renewed over 90% of contracts. The pipeline is increasing, indicating potential growth in H2. Q: What are your expectations for free cash flow and restructuring cash outflows for the second half of the year? A: Unidentified_2: We aim to maintain cash in advance at similar levels to last year, around the 300 million mark, despite not actively seeking advance payments. Restructuring cash outflows will accelerate, with most restructuring expected to be completed by summer 2026. Q: Do you expect a significant improvement in your book-to-bill ratio in H2, and how confident are you in achieving organic growth next year? A: Unidentified_1: We anticipate the book-to-bill ratio to exceed 80% and aim for around 100% by Q4. The pipeline is increasing, and we are confident in achieving organic growth next year, supported by a strong restructuring plan that will enhance profitability regardless of top-line growth. Q: Can you provide details on the renewed contract in the automotive sector and its impact on revenue and profit? A: Unidentified_1: The renewed contract with a German OEM was previously at a negative margin. We have renegotiated it to a positive margin of around 10%, improving both revenue and profitability. This reflects our strategy to address loss-making contracts. Q: Are there any expected cash outflows related to litigation or loss-making contracts in the future? A: Unidentified_1: We do not anticipate significant cash outflows from litigation in H2. For loss-making contracts, we are actively working to reduce cash outflows, with one major contract expected to stop bleeding next year, and another being negotiated for termination. For the complete transcript of the earnings call, please refer to the full earnings call transcript. This article first appeared on GuruFocus. Sign in to access your portfolio
Yahoo
02-08-2025
- Yahoo
NEWS OF THE WEEK: Phil Collins reveals reason for hospital stay
Despite speculation that the Genesis frontman has been receiving hospice care, his rep shut down concerns about his health. In a statement to People, he insisted that rumours of any terminal illness are "completely incorrect". The latest update comes six months after the drummer shared how his health issues - which included severe nerve damage from a 2007 spinal injury - had been impacting his ability to make music. "I keep thinking I should go downstairs to the studio and see what happens. But I'm not hungry for it anymore. The thing is, I've been sick, I mean very sick.".
