
What every man needs to know about diet and sperm health
Tired of too many ads? go ad free now
But there is growing evidence that advanced parental age, either maternal or paternal, is associated with increased difficulty in conceiving, along with susceptibility in children. Many studies have shown that the fertility rate declines with men's age. A 2020 study revealed that conception is 30% less likely for men older than 40 than for men younger than 30. Another study published in the Human Reproduction Update revealed that over the past 46 years, global sperm counts have dropped by more than 50%.
So, what's going on with men's reproductive health? And what role does diet play in this mounting crisis?
Decline in male fertility is not just associated with age
While aging naturally affects sperm quality, like reductions in semen volume, total sperm count, and sperm motility, lifestyle and dietary factors are also seen as powerful contributors to male infertility. One of the main culprits? Oxidative stress. This condition occurs when the body has an imbalance between free radicals and antioxidants, and it's especially harmful to sperm.
Oxidative stress causes sperm DNA fragmentation, lower motility, and reduced chances of conception.
Diet is a major determinant of oxidative stress levels. Let's discuss the food that harms male fertility:
Processed and high-fat foods:
Diets heavy in processed meats (like bacon, sausage, and hot dogs) and saturated fats (often found in fast food and fried items) are linked to lower sperm quality. These foods reduce testosterone levels and increase abnormal sperm morphology, which can impact fertility.
Tired of too many ads? go ad free now
Alcohol and tobacco:
While not foods per se, these lifestyle choices are important to mention. Alcohol interferes with sperm production and reduces libido, while smoking damages sperm DNA, reduces motility, and lowers count. When combined with a poor diet, their impact on male fertility is even more severe.
Micronutrient deficiencies:
Many men are unaware that lacking essential nutrients can silently sabotage their semen quality.
Zinc, selenium, and folate are crucial for spermatogenesis (the process of sperm production). Inadequate levels of these nutrients impair sperm development and reduce semen quality.
Tips to boost sperm quality
The good news? The male reproductive system is responsive to positive changes. But patience is key. Sperm regeneration takes about 74 days, so dietary and lifestyle modifications must be sustained for at least 2–3 months before improvements are noticeable.
Here's how to get started:
- Antioxidants:
Yes, antioxidants are your semen's best friends. Clinical studies have shown that nutrients like vitamin C, vitamin E, lycopene, and CoQ10 can improve sperm concentration and motility by fighting oxidative stress and protecting sperm DNA from damage.
- Maintain a healthy weight:
Body weight plays a crucial role in male fertility. Obesity can cause a drop in testosterone and an increase in estrogen levels in men, directly impacting the semen quality.
- Quit smoking and limit alcohol:
These habits not only harm your overall health but are directly linked to decreased sperm quality. Cutting them out can improve fertility outcomes.
Male fertility is no longer a silent subject, and it shouldn't be. If you're planning to have a baby or are concerned about your reproductive health, it's time to take a proactive approach. A semen analysis can offer vital insights into your sperm count, motility, and morphology, giving you a clear picture of where your fertility stands.
Many leading brands in the country now offer advanced semen analysis and expert consultation to guide you toward the best reproductive health.
The truth is, your daily choices, what you eat, drink, and how you live, impacts your sperm as well as the health of your future children. As science continues to uncover the powerful link between diet and sperm health, it's clear that men must take equal ownership of their reproductive wellness.
Authored by: Dr Sonu Taxak, Director & Senior IVF Consultant at Yellow Fertility
You Clean Your Diet, But What About Your Company? Sadhvi Bhagawati Saraswati's Powerful Truth

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles


Time of India
11-07-2025
- Time of India
8-year-old wheelchair-user walks again after receiving experimental drug for rare and fatal genetic condition
I magine a vibrant 8‑year‑old boy, once an avid runner and a spirited soccer player, confined to a wheelchair by a rare, fatal genetic disease that robbed him of his ability to walk. Traditional CoQ10 supplements offered no relief when his condition rapidly worsened. Then a pioneering experimental drug – 4‑hydroxybenzoate (4‑HB) – designed to bypass his genetic deficiency, not only halted his decline but restored his mobility in just weeks. Within weeks of compassionate‑use treatment approved by the FDA at NYU Langone, he walked again – hiking, go-karting, and playing with renewed vigor. This extraordinary case, the first human success for HPDL deficiency, signals a monumental shift in rare disease therapy, transforming despair into hope. It not only redefines treatment for HPDL deficiency but also shines a beacon of hope for rare mitochondrial diseases everywhere. Read on to know more. From an athlete to being wheelchair-bound : The onset of HPDL deficiency In mid-2023, this otherwise healthy boy – a competitive runner and soccer enthusiast – started showing worrying signs: stumbling, ankle stiffness, and loss of coordination. He began experiencing involuntary muscle contractions in his ankles while playing soccer. Within months, these symptoms escalated; by November, he needed a wheelchair and struggled even to stand. Genetic testing at NYU Langone revealed biallelic mutations in the HPDL gene, essential for producing coenzyme Q10 (CoQ10), a vital antioxidant for mitochondrial health. HPDL deficiency disrupts the CoQ10 biosynthesis pathway, impairing cellular energy production. Without CoQ10, neurons and muscles lose function, leading to spasticity, paralysis, seizures, and often early death, with severe forms manifesting in infancy. In this child's delayed-onset case, symptoms appeared later but progressed swiftly. The CoQ10 conundrum: Why supplements aren't enough CoQ10 is widely available as a dietary supplement and is generally safe. However, CoQ10 supplements are completely ineffective in HPDL cases affecting the brain because CoQ10 molecules are too large and hydrophobic to cross the blood–brain barrier. Without reaching the brain, supplemental CoQ10 offered minimal benefit, essentially leaving the underlying condition unchecked. While CoQ10 supplements may help peripheral tissues, they leave neurological symptoms – spasticity, paralysis, and rapid degeneration – untreated. A biochemical breakthrough: Smart 'tailor-made' solution In 2021, researchers at NYU, led by Dr. Michael Pacold, discovered that HPDL catalyzes an early step in CoQ10 synthesis: converting 4‑hydroxy‑methyladenine (4‑HMA) to 4‑hydroxybenzoate (4‑HB), a precursor that is small and hydrophilic enough can cross the blood–brain barrier. In HPDL-deficient mice, oral 4‑HB restored CoQ10 production, significantly improved neurological function, and rescued 90% of subjects when administered early. Translating this from mice to humans presented risks, but given the rapid deterioration and the absence of other options, the child's parents and medical team pursued a compassionate-use approval from the FDA. Compassionate use in humans: Life-changing progress By November 2023, with the boy's condition worsening and time running out, NYU's team – led by Dr. Claire Miller – obtained FDA approval for single-patient investigational use of 4‑HB. The treatment began in December 2023. In July 2024, the boy began daily oral doses of 4‑HB dissolved in water. Initial mood and side effects varied – he experienced nausea occasionally – but the potential benefits were undeniable. Remarkably, within just a month, he regained the ability to walk and walked more than half a mile in Central Park. His progress was profound. Over the next year, he completed a four‑mile hike and even enjoyed go-karting – all without serious side effects. Doctors report a significant restoration of motor function and stamina. The boy can complete his daily activities without assistance—though rigorous sports remain off-limits. There have been no severe side effects reported so far. Impact and perspective: A medical breakthrough This case represents the first successful application of 4‑HB therapy in a human with HPDL deficiency. Classified by experts as a "bench-to-bedside" success, it demonstrates how basic biological insights – like metabolic pathways – can be translated into transformative therapies. Pacold highlighted the value of NIH-funded foundational research: without it, 4‑HB's pathway to human use wouldn't exist. However, experts emphasize that while the results are unprecedented, caution remains essential. One patient's outcome isn't enough to define safety or efficacy broadly. Nevertheless, the experimental treatment offers proof-of-principle and has prompted plans for larger clinical trials across ages and HPDL variants. Broader implications for rare disease care This case highlights the potential of precision, mechanism-based therapies for ultra-rare genetic disorders – many of which lack approved treatments due to their complexity and small patient populations. It also illustrates how compassionate-use pathways can bridge the gap between preclinical research and patient care. For HPDL deficiency, this success sets the stage for larger clinical trials. Researchers are now exploring the treatment's wider applicability and grappling with whether 4‑HB acts solely by restoring CoQ10 or if other mechanisms are at play. A beacon of hope for rare disease communities Rare genetic disorders often lack effective therapies due to low patient numbers and complex biology. This milestone highlights how customized, mechanism-based interventions can break new ground. It reiterates the importance of NIH-funded basic science – Pacold's pathway elucidation stemmed from foundational grants. Dr. Miller reflected on how 'movement is identity and personality,' and described the turnaround as 'heartwarming.' As the boy himself reportedly asked, "When are you going to make this a pill?" – a reminder of evolving patient-driven optimism. Looking ahead What's ahead now? Next steps focus on clinical expansion: enrolling more pediatric patients, refining dosing, and assessing long-term outcomes. Scientists also aim to clarify whether 4‑HB works solely by boosting CoQ10 or also via unexpected mechanisms. Additionally, assessing whether similar treatments can address broader CoQ10 deficiencies is a promising, yet cautious, discussion. This is how Aryan Khan spent the money in jail that he received from his father Shah Rukh Khan: Reports


Time of India
03-07-2025
- Time of India
RFK Jr.'s alarming claims on sperm counts and male fertility spark debate—experts urge caution
U.S. Health and Human Services Secretary Robert F. Kennedy Jr. has ignited a nationwide debate after issuing stark warnings about declining testosterone and sperm counts among American boys and young men. Kennedy's claims—delivered in recent interviews and public appearances—have fueled doomsday predictions about a looming 'male fertility crisis,' but medical experts urge the public to look at the full scientific picture before panicking. RFK Jr.'s claims: 'An existential problem' Kennedy has repeatedly asserted that today's American teenage boys have less testosterone than a 68-year-old man and that sperm counts have plummeted by 50% over the past half-century. He links these trends to the widespread use of artificial food dyes, ultra-processed foods, and environmental chemicals, calling the issue 'existential' for the nation's future. 'A teenager today, an American teenager, has less testosterone than a 68-year-old man,' Kennedy said on Fox News. 'Testosterone levels have decreased by 50 percent from historical averages. … Sperm counts have plummeted by 50 percent, and girls are entering puberty six years sooner.' Kennedy has made these warnings a centerpiece of his public health agenda, tying them to his push for a ban on eight common artificial food dyes and stricter regulation of chemicals such as atrazine , a pesticide he describes as 'extraordinarily toxic' and linked to low sperm counts and birth defects. The science: Is there really a crisis? There is scientific evidence of a global decline in sperm counts. A widely-cited 2022 review in the journal Human Reproduction Update found that average sperm counts dropped by more than 50% worldwide between 1973 and 2018, a trend that has sparked concern among fertility specialists. Similarly, a Yale School of Medicine study published in 2020 found that testosterone levels among young American men fell from 1996 to 2020. However, experts say the reality is far more nuanced. Doctors and reproductive health researchers caution that changes in laboratory techniques and reporting standards over the decades make direct comparisons difficult. Some also note that while sperm counts may be declining, most men today still fall within the range considered fertile. Live Events 'The health secretary has warned about falling sperm counts in teens. Doctors say the reality is far more nuanced and little cause for panic.' What's driving the decline? Researchers point to a complex mix of factors: Obesity and poor diet: Linked to lower testosterone and fertility, and more prevalent among U.S. youth. Chemical exposures: Endocrine disruptors in plastics, pesticides (like atrazine), and synthetic food additives are under scrutiny for their potential impact on reproductive health. Lifestyle factors: Alcohol use, smoking, stress, and lack of exercise all play a role. Socioeconomic and environmental influences: These may also contribute to earlier puberty in girls and changes in male reproductive health. Kennedy's statements have drawn both support and criticism. Some see his focus on food and chemical safety as overdue, while others accuse him of spreading panic and making unsubstantiated medical claims. Notably, Kennedy has also made controversial remarks on autism and other health issues, drawing further scrutiny from scientists and public health officials.


News18
04-06-2025
- News18
This Cheap Tablet May Help Prevent Heart Failure, Says Study
Last Updated: Researchers found the drug reduced heart stiffness caused by diabetes, which weakens energy conversion in the heart and raises the risk of sudden heart failure A new study has revealed that an inexpensive medication can significantly reduce the risk of heart failure or heart attack in diabetes patients. According to the study, diabetes patients who took the affordable drug Mitoquinone had a much lower risk of heart attack compared to those who did not take the medication. Priced at just 90 pence in Britain, Mitoquinone is available as an over-the-counter supplement in both the UK and the US. According to a Daily Mail report, the most remarkable finding of the study was that heart damage in diabetic patients was also reversed after being given the drug mitoquinone. This is significant, as heart muscles typically weaken in diabetic patients. Mitoquinone is a synthetic antioxidant that protects cells and is a modified form of CoQ10, an antioxidant naturally found in the body. Study author and heart specialist at the University of Leeds, Dr. Henry Proctor, called the results encouraging. He added that if the study had been conducted over a longer period, the difference between the mitoquinone and non-mitoquinone groups might have been more apparent in serious indicators of heart health. However, he expressed hope that larger studies will continue on the benefits of this antioxidant, which could one day help diabetic patients prevent heart failure in advance. Heart Function Rises 15% With New Drug The results of the study were presented at the British Cardiovascular Society conference in Manchester. Half of the patients included in the study were given 40 mg of mitoquinone daily along with their usual diabetes treatment, while the rest were given only their usual medicines. The heart health of both groups was studied and re-evaluated after four months. At the end of the trial, the heart function of the group taking mitoquinone was found to be 15 percent higher than at the beginning of the study. The mitoquinone group also showed improvement in exercise tests. Their hearts returned to a relaxed state more easily after exercise. Researchers said that after taking the medicine, the stress and stiffness in the heart caused by diabetes was reduced. Diabetes damages the heart in such a way that the heart becomes weak in converting energy. Over time, this obstacle increases the stress and damage to the heart muscle cells, due to which the heart muscles become stiff. In this condition, more effort is needed to pump blood throughout the body, which increases the risk of sudden heart failure. No Side Effects Reported Mitoquinone helps protect cells from damage. It is already sold online as a health supplement, with a 60-tablet bottle priced around £50—roughly 86 pence per tablet. However, these over-the-counter doses are much lower than those used in the study. Importantly, no side effects were reported in the group taking mitoquinone. Scientists are now planning a larger trial involving more patients to strengthen their findings. If future studies confirm these results, experts hope mitoquinone could one day be used to protect the hearts of diabetic patients for longer. First Published: June 04, 2025, 18:22 IST