ISRO signs MoU with NIOT for submersible spherical vessel MATSYA-6000
's
Vikram Sarabhai Space Centre
(VSSC) has signed a Memorandum of Understanding with the
National Institute of Ocean Technology
(
NIOT
) to develop a
submersible spherical vessel
,
MATSYA-6000
, which can carry humans into the sea up to a depth of 6km.
The
human-occupied vehicle
(HOV) is part of 'Samudrayaan', a project under the
Deep Ocean Mission
of the Ministry of Earth Sciences (MoES), according to ISRO.
In a release issued by the space agency on Wednesday, the design of the HOV along with development of the material for realisation was carried out by VSSC.
by Taboola
by Taboola
Sponsored Links
Sponsored Links
Promoted Links
Promoted Links
You May Like
We Can't Believe She Wore That To The Red Carpet
Golfhooked
Read More
Undo
The sphere measuring 2,260mm diameter with a wall thickness of 80mm, is made out of Titanium alloy and is expected to withstand an external pressure up to 600bar under low temperature conditions of -3°C.
It has the capability to carry a three-member crew into the deep ocean, the release added.
Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles
Time of India
36 minutes ago
- Time of India
Death of 5 billion sea stars mystery solved: Scientists reveal what killed the iconic star-shaped creatures
Scientists say they have finally discovered the cause behind the death of more than 5 billion sea stars along the Pacific coast of North America, solving a mystery that has lasted over a decade. As per an AP report, starting in 2013, a mysterious disease called sea star wasting disease led to a massive die-off of sea stars (often called starfish), from Mexico to Alaska. More than 20 species were affected, with the sunflower sea star hit hardest, losing nearly 90% of its population in just five years. Productivity Tool Zero to Hero in Microsoft Excel: Complete Excel guide By Metla Sudha Sekhar View Program Finance Introduction to Technical Analysis & Candlestick Theory By Dinesh Nagpal View Program Finance Financial Literacy i e Lets Crack the Billionaire Code By CA Rahul Gupta View Program Digital Marketing Digital Marketing Masterclass by Neil Patel By Neil Patel View Program Finance Technical Analysis Demystified- A Complete Guide to Trading By Kunal Patel View Program Productivity Tool Excel Essentials to Expert: Your Complete Guide By Study at home View Program Artificial Intelligence AI For Business Professionals Batch 2 By Ansh Mehra View Program 'It's really quite gruesome,' said Alyssa Gehman, a marine disease expert at the Hakai Institute in British Columbia, who worked on the new research. 'Healthy sea stars have puffy arms sticking straight out, but with the disease, they develop lesions and then their arms actually fall off.' by Taboola by Taboola Sponsored Links Sponsored Links Promoted Links Promoted Links You May Like This Could Be the Best Time to Trade Gold in 5 Years IC Markets Learn More The cause? A bacteria known as Vibrio pectenicida , the same type that also affects shellfish, was identified in a new study published in the journal Nature Ecology and Evolution. 'This solves a long-standing question about a very serious disease in the ocean,' said Rebecca Vega Thurber, a marine microbiologist at the University of California, Santa Barbara, who was not part of the study. Live Events For years, scientists believed the cause might be a virus, particularly a densovirus. But further research revealed that this virus exists in healthy sea stars too, and wasn't responsible for the illness. Researchers had also missed the real cause earlier because they mainly studied dead sea stars, which no longer had the internal fluid needed for proper analysis. This time, scientists focused on coelomic fluid, the liquid inside living sea stars, and found the harmful bacteria there. 'It's incredibly difficult to trace the source of environmental diseases , especially underwater,' said microbiologist Blake Ushijima from the University of North Carolina, who wasn't involved in the study. He called the research 'really smart and significant.' Now that the cause is known, scientists believe they can start efforts to protect and restore sea star populations. According to Melanie Prentice, co-author of the study, researchers can now test which sea stars are still healthy, explore breeding in captivity, and move healthy individuals to areas where the population has collapsed. They may also test whether some sea stars have natural immunity, and whether probiotics could help protect them. This work is essential not just for the sea stars, but for the entire marine ecosystem. Sunflower sea stars are known for eating sea urchins, which helps keep their numbers in check. Since the sea stars have vanished, sea urchins have taken over, destroying about 95% of kelp forests in Northern California within ten years. These kelp forests are often called the 'rainforests of the ocean,' and they provide food and shelter for fish, sea otters, seals, and many other marine species. 'Sunflower sea stars look sort of innocent when you see them,' said Gehman, 'but they eat almost everything that lives on the bottom of the ocean. They're voracious eaters.' Now, with this new breakthrough, scientists hope to bring sea star numbers back, and help restore the Pacific's kelp forests. Inputs from AP
Time of India
an hour ago
- Time of India
Late puberty in boys may increase risk of type 2 diabetes: Study finds
Puberty marks a critical phase of growth, shaping not just physical and hormonal development but also future health. While early puberty has long been associated with certain risks, new research suggests that late puberty in boys may also have serious implications, particularly a higher chance of developing type 2 diabetes in adulthood. A large-scale study from Israel has found that boys who go through puberty later than average are significantly more likely to be diagnosed with type 2 diabetes later in life. These findings point to puberty timing as a potential early marker for chronic disease risk in young males. Study finds delayed puberty linked to higher risk of type 2 diabetes in adulthood The study , conducted in Israel, analysed health data from over 960,000 adolescent boys aged between 16 and 19 years who were assessed for military service between 1992 and 2015. Of these, more than 4,300 were found to have experienced delayed puberty. Researchers followed these individuals until 2019, monitoring the development of type 2 diabetes over time. The findings revealed that boys with delayed puberty were approximately 2.5 times more likely to develop type 2 diabetes in early adulthood than those whose puberty occurred at the expected time. Even after accounting for other influencing factors such as birth year, socioeconomic background, education level, region of origin, and cognitive ability, the association remained strong. by Taboola by Taboola Sponsored Links Sponsored Links Promoted Links Promoted Links You May Like Free P2,000 GCash eGift UnionBank Credit Card Undo Notably, when researchers adjusted for body mass index (BMI), a well-known contributor to diabetes, the risk was still 37% higher among those with delayed puberty. This suggests that puberty timing itself may play an independent role in shaping future metabolic health. The difference in diabetes development between the two groups was significant: 140 per 100,000 boys with delayed puberty developed type 2 diabetes each year. In contrast, only 41 per 100,000 boys with normal pubertal timing developed the condition annually. These figures suggest that puberty timing may serve as a valuable predictor of future health risks. How puberty timing in boys affects long-term health and diabetes risk Puberty is a period of intense hormonal and physical transformation. During this stage, the body undergoes significant changes in insulin sensitivity, fat distribution, and hormone levels. Researchers believe that there may be a sensitive developmental window during adolescence when these hormonal shifts have lasting effects on how the body processes glucose and stores fat. Just as early childhood development influences cognitive and language skills, puberty could shape the body's long-term metabolic capacity. Delayed puberty may reflect underlying hormonal imbalances or disruptions in growth signals, which could contribute to a higher risk of insulin resistance, a key factor in the development of type 2 diabetes. This aligns with other research suggesting that adolescence is a 'programming' phase for the endocrine system, influencing not only reproductive health but also overall metabolic function. Rising type 2 diabetes in teens and young adults: How puberty timing may play a role Traditionally considered a disease of older adults, type 2 diabetes is now increasingly being diagnosed in teenagers and young adults. Factors such as sedentary lifestyles, high-calorie diets, and obesity are major contributors to this trend. However, the new research adds an important layer: developmental timing, particularly the onset of puberty, may also influence risk, independent of lifestyle choices. Type 2 diabetes occurs when the body becomes resistant to insulin or does not produce enough of it, leading to elevated blood sugar levels. Over time, this condition can result in complications such as cardiovascular disease, kidney damage, nerve dysfunction, and vision problems. Recognising early-life signals like delayed puberty could therefore help identify individuals at greater risk before these complications arise. Delayed puberty in boys could be an early warning sign for future health risks In most cases, late puberty in boys is viewed as a normal variation with no lasting consequences. However, the latest findings challenge this view, suggesting that it may be a valuable clinical marker for identifying future health risks. Adolescents with delayed puberty may benefit from closer health monitoring, particularly in terms of blood sugar levels, weight management, and hormonal evaluation. If a child shows signs of delayed puberty, parents and healthcare providers should not dismiss it lightly. Instead, they should consider further assessment to understand the underlying cause and explore whether additional health checks are warranted. This is particularly important for boys with a family history of diabetes or other metabolic disorders. Lifestyle tips to reduce diabetes risk in boys with delayed puberty For boys with delayed puberty or a family history of diabetes, early lifestyle changes can make a difference. Recommended actions: Encourage regular physical activity, such as walking, cycling, or sports. Limit processed foods, sugary snacks, and sugary drinks. Promote a balanced diet rich in whole grains, lean protein, fruits, and vegetables. Ensure sufficient sleep and manage stress. Even small changes made consistently can help reduce the risk of diabetes and support healthy development during adolescence. This research sheds new light on puberty as more than just a developmental milestone; it may also signal long-term health risks like type 2 diabetes. Recognising delayed puberty as a potential early warning sign allows for earlier monitoring and intervention. As diabetes rises among younger populations, identifying such risk markers is key to prevention. With timely support and healthy lifestyle changes, at-risk adolescents can be guided towards a healthier future. Also Read: Side effects of eating non-veg daily: Study reveals 9 long-term health risks
Time of India
an hour ago
- Time of India
Alzheimer's Disease: Scientists find potential remedy to the progressive brain disorder (in a common chemical element)
Alzheimer's disease affects millions of people around the world, slowly damaging memory, thinking, and behavior. For decades, scientists have searched for a way to stop or even slow down this heartbreaking condition. Now, a promising discovery has brought new hope, and it comes from an unexpected source: a common chemical element. Read on to know more about the groundbreaking discovery. What is Alzheimer's Disease? Alzheimer's disease is a progressive brain disorder that slowly destroys memory and thinking skills. In its later stages, it can make simple daily tasks, like eating or recognizing loved ones, nearly impossible. It is the most common type of dementia, a general term for memory loss and cognitive decline. According to the World Health Organization, more than 55 million people worldwide live with dementia, and Alzheimer's accounts for 60-70% of those cases. The exact cause of Alzheimer's is not fully understood, but it's linked to the build-up of toxic proteins in the brain, including amyloid plaques and tau tangles, which interfere with normal brain function. Lithium: The missing link In a discovery that's been nearly 10 years in the making, researchers at Harvard Medical School have uncovered a surprising new role for lithium, a metal best known for treating bipolar disorder. by Taboola by Taboola Sponsored Links Sponsored Links Promoted Links Promoted Links You May Like If A Cat Bites Their Owner Heres What It Really Means Gloriousa Undo This new research shows that lithium is not only naturally present in the body in tiny amounts, but it also plays a key role in keeping our brains healthy as we age. The study, published in the journal Nature , found that even a small drop in natural lithium levels in the brain can lead to changes linked to Alzheimer's disease and brain aging. While more research is needed, scientists are hopeful that this could lead to new ways to prevent or slow down the disease. Now, lithium has been used as a mood stabilizer for over a century and was officially approved by the FDA in 1970. But this new study shows that even tiny, natural levels of lithium in the body, similar to vitamins and minerals, are important for normal cell function. 'It is a potential candidate for a common mechanism leading to the multisystem degeneration of the brain that precedes dementia,' said Dr. Bruce Yankner, senior author of the study, as reported by CNN. 'It will take a lot more science to determine whether this is a common pathway… or one of several pathways,' he added. 'The data are very intriguing.' What did the study find? Researchers at Harvard and Rush University fed mice a diet with reduced lithium. They discovered that: Healthy mice developed brain inflammation and signs of accelerated aging. Mice bred to develop Alzheimer's-like symptoms showed faster buildup of harmful brain plaques and more memory loss. Keeping normal lithium levels helped protect the mice's brains. The team also tested a special form of lithium called lithium orotate, which doesn't bind to harmful proteins in the brain. Mice treated with lithium orotate saw a reduction in brain plaques and memory issues, even reversing some Alzheimer's-like symptoms. These mice were able to complete memory tasks that untreated mice could not. Can lithium help the brain? A key finding from the study was that beta amyloid plaques, the sticky clumps seen in Alzheimer's patients, bind to lithium in the brain, including the lithium naturally present in our bodies. This binding traps the lithium, making it unavailable to nearby brain cells, especially microglia, which are cells that clean up waste in the brain. Without enough lithium, microglia couldn't clear out the beta amyloid, allowing the plaques to build up and cause more damage. Yankner described this as a 'downward spiral;' the more plaques there are, the more lithium they absorb, and the harder it is for the brain to fight back. Where do we get natural lithium? Lithium exists naturally in water, soil, and food. The study found that people with Alzheimer's had lower levels of lithium in their brain tissue, while those with normal memory function had higher levels. Foods high in natural lithium include: Leafy greens Nuts Legumes Spices like turmeric and cumin Some mineral waters This may help explain why diets rich in these foods, like the Mediterranean diet, are often linked to lower dementia risk. More supporting evidence This isn't the first time lithium has been linked to brain health: A 2017 study in Denmark found that people who drank lithium-rich tap water were less likely to develop dementia. A 2022 study in the UK found that people prescribed lithium had half the risk of Alzheimer's compared to those who weren't. Until now, lithium has been mostly seen as a drug. But this new research suggests it may also be a natural nutrient essential to normal brain function, one that we're only just beginning to understand. What's next? Researchers are now studying why lithium levels drop in the brain as people age. It may involve: Reduced intake from diet Genetic factors Changes in how the brain absorbs lithium The findings were made possible by new technology that allowed scientists to measure extremely small amounts of lithium, something that wasn't possible before. 'This is one of those moments in science where something unexpected opens the door to an entirely new understanding,' said Yankner. 'We still have much to learn, but we now know lithium plays a bigger role in brain health than we ever imagined.' Word of caution Even though the results are exciting, researchers warn against self-treatment. Prescription lithium doses are much higher than what's naturally found in the body and can cause kidney or thyroid damage if taken without medical supervision. 'The lithium treatment data we have is in mice, and it needs to be replicated in humans,' Yankner explained. 'A mouse is not a human. Nobody should take anything based just on mouse studies.' Still, early signs are promising. The low-dose lithium used in the study showed no signs of toxicity in mice. Yankner hopes human safety trials will begin soon. This article is for informational purposes only and does not constitute medical advice. The research discussed involves early-stage findings primarily based on animal studies. Lithium supplements or treatments should not be taken without medical supervision, as incorrect usage can pose serious health risks. Always consult a qualified healthcare professional before making changes to your medication or dietary routine. Researchers discover toxic process involving 'jumping genes'
