
Digital Brain Fasting: All About The Detox Your Brain Desperately Needs
In a digital savvy world, the mind is consuming content digitally at an increased pace. This can lead to digital overexposure that can impact your brain health.
In today's hyper-connected world, neurologists are witnessing an alarming surge in patients, especially young professionals, complaining of persistent headaches, neck pain, memory issues, and sleep disturbances. The underlying cause in many of these cases isn't a neurological disease, but something far more pervasive: digital overexposure. From workplace screens to bedtime scrolling, the digital deluge is silently taking a toll on our brains.
'Many of my patients are in their 20s and 30s. They're software engineers or corporate professionals logging 8–10 hours in front of screens daily, and then they go home and continue using phones or tablets," shares Dr. Praveen Sharma, Consultant – Neurology, Apollo Speciality Hospital, Jayanagar, Bengaluru. It's a pattern that's contributing to a new kind of cognitive burnout – one that demands a radical reset: digital brain fasting.
Extended screen time, especially late into the night, interferes with the brain's melatonin production, which is essential for restful sleep. The result? Fatigue, irritability, and foggy memory.
'Poor posture and prolonged device use can cause cervical or lumbar spondylosis, carpal tunnel syndrome, and even arthritis," explains Dr. Sharma. He warns that beyond the physical, there are significant mental health implications too: 'Negative content, online bullying, and lack of face-to-face interaction are contributing to anxiety, depression, and even suicide ideation," he explains.
Gaming addiction is particularly concerning among teens. 'Many lose touch with reality, developing aggressive tendencies and isolation," he adds.
What Does a Digital Detox Actually Look Like?
Step one is awareness. 'The brain gets a dopamine hit from reels and scrolls – making digital habits hard to break," notes Dr. Sharma. But breaking them is critical.
Replace screen time with healthier dopamine triggers: books, physical activity, hobbies, and real-world social interaction. He suggests simple rules: no screen time an hour before bed, putting phones on silent during family meals, and leading by example – especially for parents.
'If screens are unavoidable, reduce brightness, keep screens at eye level, and stretch every two hours," Dr Sharma advises.
The Long-Term Benefits of Digital Brain Fasting
Digital fasting isn't just about disconnecting; it's about rewiring our brains to function better. 'It improves memory, reduces mood swings, builds self-confidence, and lowers risks of neurological issues like migraines, spondylosis, and even Alzheimer's," Dr. Sharma emphasises.
Digital engagement isn't inherently harmful. He acknowledges, 'Puzzles or reading on phones is fine – if it's within limits."
The real danger lies in passive, addictive consumption driven by the fear of missing out.
view comments
First Published:
August 06, 2025, 13:57 IST
Disclaimer: Comments reflect users' views, not News18's. Please keep discussions respectful and constructive. Abusive, defamatory, or illegal comments will be removed. News18 may disable any comment at its discretion. By posting, you agree to our Terms of Use and Privacy Policy.

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles


Hindustan Times
8 hours ago
- Hindustan Times
Scientists Are Finally Making Progress Against Alzheimer's
Buck up, baby boomers. Sixty may not be the new 40, but 80 could soon be the new 60. A trove of new Alzheimer's research suggests that medicines and lifestyle changes can not only slow but prevent, even reverse, the debilitating disease. Alzheimer's is characterized by a buildup of amyloid plaque and tangles of tau proteins in the brain. They trigger inflammation, which in turn causes cognitive decline and neuron death. Scientists don't know exactly what causes this destructive cascade. The disease's complexity stymied the quest for treatments for decades, but no more. Scientists are learning quickly. The only two approved treatments for Alzheimer's that can slow progression—developed by Eli Lilly and Biogen-Eisai—work by removing amyloid plaque. They reduce cognitive decline by 25% to 36% over 18 months compared with a placebo group in clinical trials. Those in the earliest stages of the diseases saw bigger benefits, and nearly half of those in the Lilly trial showed no progression after a year. Some back-bench scientists who don't treat Alzheimer's patients dismiss such benefits as not meaningful. But the medications could mean more years when your mother recognizes you. Who wouldn't find that meaningful? Recent follow-up studies have also shown that the benefits grow over time—doubling over three years for Eli Lilly's treatment and quadrupling over four years for Biogen-Eisai's. Drugmakers are studying whether eliminating amyloid can ward off Alzheimer's in preclinical patients—those without cognitive symptoms—as statins can prevent cardiovascular disease. Lifestyle changes can augment the benefits. A recently published randomized trial found that a 'treatment' of regular physical and cognitive exercise, nutritious diet and social engagement improved cognitive performance among previously sedentary older adults at risk of dementia. A smaller study earlier this summer found such lifestyle interventions improved metal acuity of early-stage Alzheimer's patients, as well as physiological measures of brain health. The treatment group in the latter study also showed an increase in beneficial bacteria in their guts. Why is this notable? Studies have found that microbiomes of Alzheimer's patients differ from those of healthy seniors. For one, Alzheimer's patients have lower levels of Akkermansia muciniphila, a bacterial strain that produces the short-chain fatty acid propionate from dietary fiber. Propionate helps regulate appetite as well as brain inflammation and amyloid buildup. In one study, propionate was found to reduce brain inflammation and amyloid in mice with the rodent version of Alzheimer's disease. So perhaps a probiotic cocktail or more fiber in the diet could slow progression, much like antiamyloid treatments. Another potential antidote: lithium salts. A study published last week in the journal Nature found that amyloid can trap lithium, resulting in a deficiency of the mineral. Lithium protects neurons. Mice given a lithium-deficient diet had more amyloid, damaged neurons and impaired learning and memory. A low dose of lithium orotate reversed cognitive problems and restored memory. How the microbiome and lithium affect Alzheimer's is the sort of basic scientific research that deserves more government attention and funding. Ditto the effects of viruses and vaccines. Take herpes viruses, which cause chickenpox, shingles and cold sores. Most seniors have been infected by at least one type of herpes virus. After an initial infection, the virus lies dormant in cells, including in the brain. Reactivation of the virus has been linked to increased amyloid buildup, brain inflammation and risk of dementia. Vaccines and antivirals may be able to help prevent Alzheimer's. A quasirandomized control trial earlier this year found that people vaccinated for shingles were 20% less likely to develop dementia. Recent research has also found that head injuries increase amyloid and brain inflammation by reactivating herpes viruses. So perhaps administering herpes antivirals after concussions could help protect the brain. Alzheimer's tends to run in families, and the APOE4 variant is the biggest genetic risk factor. Women with one copy, inherited from mom or dad, are at four times as much risk. Those with two, one from each parent, have 10 times the risk. Men with one variant show no increased risk, and those with two copies are at fourfold increased risk. You can't change your genes, but an experimental cholesterol drug was recently found to reduce Alzheimer's hallmarks in APOE4 carriers in a late-stage trial. Perhaps that's because APOE4 can cause cholesterol to build up in the brain. All these studies underline the potential to stop Alzheimer's dead in its tracks—as long as the Food and Drug Administration doesn't get in the way. The Health and Human Services Department on Saturday announced that Vinay Prasad was returning as the FDA head of biologics and gene therapies. Dr. Prasad stepped down late last month after a public backlash over his scuttling of a gene therapy for Duchenne muscular dystrophy. He's shown similar hostility to Alzheimer's treatments. In a tweet last summer referencing Biogen-Eisai's antiamyloid treatment, he wrote that paying 'someone to come to an Alzheimer's patients house and help with the dishes' would do more good than 'funding these garbage drugs.' Tell that to the poor souls afflicted by Alzheimer's and their families who have years more of quality life together.


Mint
8 hours ago
- Mint
Scientists are finally making progress against Alzheimer's
Buck up, baby boomers. Sixty may not be the new 40, but 80 could soon be the new 60. A trove of new Alzheimer's research suggests that medicines and lifestyle changes can not only slow but prevent, even reverse, the debilitating disease. Alzheimer's is characterized by a buildup of amyloid plaque and tangles of tau proteins in the brain. They trigger inflammation, which in turn causes cognitive decline and neuron death. Scientists don't know exactly what causes this destructive cascade. The disease's complexity stymied the quest for treatments for decades, but no more. Scientists are learning quickly. The only two approved treatments for Alzheimer's that can slow progression—developed by Eli Lilly and Biogen-Eisai—work by removing amyloid plaque. They reduce cognitive decline by 25% to 36% over 18 months compared with a placebo group in clinical trials. Those in the earliest stages of the diseases saw bigger benefits, and nearly half of those in the Lilly trial showed no progression after a year. Some back-bench scientists who don't treat Alzheimer's patients dismiss such benefits as not meaningful. But the medications could mean more years when your mother recognizes you. Who wouldn't find that meaningful? Recent follow-up studies have also shown that the benefits grow over time—doubling over three years for Eli Lilly's treatment and quadrupling over four years for Biogen-Eisai's. Drugmakers are studying whether eliminating amyloid can ward off Alzheimer's in preclinical patients—those without cognitive symptoms—as statins can prevent cardiovascular disease. Lifestyle changes can augment the benefits. A recently published randomized trial found that a 'treatment" of regular physical and cognitive exercise, nutritious diet and social engagement improved cognitive performance among previously sedentary older adults at risk of dementia. A smaller study earlier this summer found such lifestyle interventions improved metal acuity of early-stage Alzheimer's patients, as well as physiological measures of brain health. The treatment group in the latter study also showed an increase in beneficial bacteria in their guts. Why is this notable? Studies have found that microbiomes of Alzheimer's patients differ from those of healthy seniors. For one, Alzheimer's patients have lower levels of Akkermansia muciniphila, a bacterial strain that produces the short-chain fatty acid propionate from dietary fiber. Propionate helps regulate appetite as well as brain inflammation and amyloid buildup. In one study, propionate was found to reduce brain inflammation and amyloid in mice with the rodent version of Alzheimer's disease. So perhaps a probiotic cocktail or more fiber in the diet could slow progression, much like antiamyloid treatments. Another potential antidote: lithium salts. A study published last week in the journal Nature found that amyloid can trap lithium, resulting in a deficiency of the mineral. Lithium protects neurons. Mice given a lithium-deficient diet had more amyloid, damaged neurons and impaired learning and memory. A low dose of lithium orotate reversed cognitive problems and restored memory. How the microbiome and lithium affect Alzheimer's is the sort of basic scientific research that deserves more government attention and funding. Ditto the effects of viruses and vaccines. Take herpes viruses, which cause chickenpox, shingles and cold sores. Most seniors have been infected by at least one type of herpes virus. After an initial infection, the virus lies dormant in cells, including in the brain. Reactivation of the virus has been linked to increased amyloid buildup, brain inflammation and risk of dementia. Vaccines and antivirals may be able to help prevent Alzheimer's. A quasirandomized control trial earlier this year found that people vaccinated for shingles were 20% less likely to develop dementia. Recent research has also found that head injuries increase amyloid and brain inflammation by reactivating herpes viruses. So perhaps administering herpes antivirals after concussions could help protect the brain. Alzheimer's tends to run in families, and the APOE4 variant is the biggest genetic risk factor. Women with one copy, inherited from mom or dad, are at four times as much risk. Those with two, one from each parent, have 10 times the risk. Men with one variant show no increased risk, and those with two copies are at fourfold increased risk. You can't change your genes, but an experimental cholesterol drug was recently found to reduce Alzheimer's hallmarks in APOE4 carriers in a late-stage trial. Perhaps that's because APOE4 can cause cholesterol to build up in the brain. All these studies underline the potential to stop Alzheimer's dead in its tracks—as long as the Food and Drug Administration doesn't get in the way. The Health and Human Services Department on Saturday announced that Vinay Prasad was returning as the FDA head of biologics and gene therapies. Dr. Prasad stepped down late last month after a public backlash over his scuttling of a gene therapy for Duchenne muscular dystrophy. He's shown similar hostility to Alzheimer's treatments. In a tweet last summer referencing Biogen-Eisai's antiamyloid treatment, he wrote that paying 'someone to come to an Alzheimer's patients house and help with the dishes" would do more good than 'funding these garbage drugs." Tell that to the poor souls afflicted by Alzheimer's and their families who have years more of quality life together.


Time of India
a day ago
- Time of India
Govt relaxes RGHS limits on OPD, tests for pensioners
Jaipur: Chief Minister Bhajan Lal Sharma Sunday approved amendments to Rajasthan Government Health Scheme (RGHS) under Rajasthan State Pensioners Medical Concession Scheme-2021, giving pensioners increased financial allowance for OPD medicines and diagnostic tests. "With this decision, we are ensuring that no pensioner faces financial hurdles in accessing essential healthcare. Our govt is committed to making medical services more accessible and responsive," CM Sharma said. As per a finance department notification, powers to relax the annual limits—earlier vested with the finance department—have now been transferred to medical & health department and Rajasthan State Health Assurance Agency (RSHA). Under the revised system, the annual Rs 50,000 limit for OPD medicines can be increased up to Rs 2 lakh by RSHA's additional CEO or joint CEO (authorised by the medical & health department). For amounts between Rs 2 lakh and Rs 7 lakh, the RSHA CEO will have the authority, and for amounts beyond Rs 7 lakh, the medical & health department will take the decision. Likewise, the Rs 5,000 limit for diagnostic tests can be enhanced by the RSHA CEO. by Taboola by Taboola Sponsored Links Sponsored Links Promoted Links Promoted Links You May Like Richard Dean Anderson Is Almost 80, See Him Now Cash Roadster Undo Pensioners will be able to apply online for such relaxations through the RGHS portal. "This step will cut red tape and save precious time for our senior citizens. Medical needs cannot wait, and now the process to increase financial limits for treatment will be faster and simpler," a senior health department official said. The move is expected to benefit thousands of pensioners across Rajasthan, ensuring timely medical care without bureaucratic delays. Stay updated with the latest local news from your city on Times of India (TOI). Check upcoming bank holidays , public holidays , and current gold rates and silver prices in your area.