‘Goblin prince': New monstersaur discovered in Utah reveals hidden secrets of Gila monster relatives
When Hank Woolley, a paleontological researcher who specializes in lizard evolution, opened a jar of bones labeled 'lizard' at the Natural History Museum of Utah, he said his first thought was, 'Oh wow, there's a fragmentary skeleton here.'
'We know very little about large-bodied lizards from the Kaiparowits Formation in Grand Staircase-Escalante National Monument in Utah, so I knew this was significant right away,' said Woolley, from the Natural History Museum of Los Angeles County's Dinosaur Institute.
With Woolley's expertise, that jar of bones — even though it was collected back in 2005 — helped lead to a new discovery of a 'racoon-sized armored monstersaurian,' a giant relative of the Gila monster, according to an announcement issued last week by the University of Utah.
Its name, Bolg amondol, was inspired by a 'goblin prince' villain in J.R.R. Tolkien's 'Lord of the Rings' universe. Bolg now represents an 'evolutionary lineage that sprouted within a group of large-bodied lizards called monstersaurs that still roam the deserts from which Bolg was recovered,' according to the U.
Woolley knew that a new species of monstersaur called for an appropriate name from an 'iconic monster creator': Tolkien, the university said in its release.
'Bolg is a great sounding name. It's a goblin prince from 'The Hobbit,' and I think of these lizards as goblin-like, especially looking at their skulls,' Woolley said.
He also used Tolkien's fictional Elvish language Sindarin to craft the species' epithet. 'Amon' means 'mound,' and 'dol' means 'head,' a reference to mound-like osteoderms (or bony deposits that act as a form of armor) found on Bolg's and other monstersaurs' skulls.
''Mound-headed Bolg' would fit right in with the goblins — and it's revealing quite a bit about monstersaurs,' the U. said.
The research published June 17 in the journal Royal Society Open Science, led by the Dinosaur Institute in Los Angeles County and the Natural History Museum of Utah, 'reveals hidden treasures awaiting future paleontologists in the bowels of museum fossil collections,' the university said, along with 'the vast potential of paleontological heritage preserved in Grand Staircase-Escalante National Monument and other public lands.'
The field collection of the specimens were conducted under paleontological permits issued by the Bureau of Land Management, which also helped fund the study with a National Science Foundation award.
SUBSCRIBE: GET THE MORNING HEADLINES DELIVERED TO YOUR INBOX
Though the Bolg specimen was first unearthed more than a decade ago, in 2005, Woolley's expertise in lizard paleontology helped determine its significance. He was the lead author of the research.
'Bolg is a great example of the importance of natural history museum collections,' said co-author Randy Irmis, an associate professor at the U. and curator of paleontology at the Natural History Museum of Utah, in the news release. 'Although we knew the specimen was significant when it was discovered back in 2005, it took a specialist in lizard evolution like Hank to truly recognize its scientific importance and take on the task of researching and scientifically describing this new species.'
Though museum collection spaces are 'sometimes stereotyped as dusty, forgotten places, the truth is quite the opposite,' the Natural History Museum of Utah said in a post about Bolg last week. The museum's paleontology collection is a 'glorified storage unit. Instead, it's a space where staff, students, volunteers, and visiting researchers can care for fossils and conduct new research.'
'It was this reevaluation of collection specimens,' the museum added, 'that led to Woolley's breakthrough research on Bolg amondol.'
Irmis said discovering a new species of lizard that's an ancestor of the modern-day Gila monster is 'pretty cool in and of itself, but what's particularly exciting is what it tells us about the unique 76-million-year-old ecosystem it lived in.'
'The fact that Bolg co-existed with several other large lizard species indicates that this was a stable and productive ecosystem where these animals were taking advantage of a wide variety of prey and different micro-habitats,' Irmis said.
Researchers identified the new species from a collection of skull, limbs, vertebrae and bony armor called osteoderms. Most fossil lizards from the dinosaur age 'are even scrappier — often just single, isolated bones or teeth — so despite their fragmentary nature, the parts of Bolg's skeleton that survived contain a stunning amount of information,' the U. said.
'That means more characteristics are available for us to assess and compare to similar-looking lizards,' Woolley said. 'Importantly, we can use those characteristics to understand this animal's evolutionary relationships and test hypotheses about where it fits on the lizard tree of life.'
The clade (or ancestral grouping) of anguimorph lizards known as monstersauria are characterized by their large size and distinctive features, like 'sharp, spire-like teeth and pitted, polygonal armor attached to their skulls.'
Bolg, the U. said, 'would have been a bit of a monster to our eyes.'
'Three feet tip to tail, maybe even bigger than that, depending on the length of the tail and torso,' Woolley said. 'So, by modern lizard standards they're a very large animal, similar in size to a Savannah monitor lizard; something that you wouldn't want to mess around with.'
Monstersauria have a roughly 100-million-year history, but their fossil record is largely incomplete, meaning Bolg's discovery is a 'big deal' to help fully understand the prehistoric lizards and their world, the university said.
'Bolg's closest known relative hails from the other side of the planet in the Gobi Desert of Asia,' the U. said. 'Though dinosaurs have long been known to have traveled between the once-connected continents of the Late Cretaceous Period, Bolg reveals that smaller animals also made the trek, suggesting there were common patterns of biogeography across terrestrial vertebrates during this time.'
Bolg was discovered in the rocks of the Kaiparowits Formation of Grand Staircase-Escalante National Monument — a formation that has emerged as a paleontological hotspot over the past 25 years, according to the U.
Those rocks have produced 'one of the most astounding dinosaur-dominated records in North America,' the university said. 'Discoveries like this underscore the importance of preserving public lands in the Western U.S. for science and research.'
Co-author Joe Sertich, from the Smithsonian Tropical Research Institute and Colorado State University, said in the release that the 'exceptional record of big lizards' from Grand Staircase-Escalante 'may prove to be a normal part of dinosaur-dominated ecosystems from North America.'
He said those lizards appear to have filled 'key roles as smaller predators hunting down eggs and small animals in the forests of Laramidia,' which is an island continent that existed during the Late Cretaceous period.
SUPPORT: YOU MAKE OUR WORK POSSIBLE
Hashtags

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles


Gizmodo
a day ago
- Gizmodo
Hibernation's Hidden Healing ‘Superpowers' Could Be Locked in Our DNA
After spending months without eating, drinking, or moving, hibernating mammals must rebound from extreme physiological changes. Two new studies suggest that the genetic 'superpowers' underlying this incredible resilience may also be present in the human genome. For these studies, published Thursday, July 31, in the journal Science, researchers at the University of Utah honed in on the specific DNA regions that help hibernators rapidly recover from muscle atrophy, insulin resistance, and brain damage. They found strong evidence to suggest that the human genome shares these genetic regions, which function as control switches for hibernator adaptations. Finding and harnessing them could lead to new treatments for type 2 diabetes, Alzheimer's disease, and other disorders, the researchers say. 'Humans already have the genetic framework,' said Susan Steinwand, a neurobiology and anatomy researcher at U of U Health and first author of one of the studies. 'We just need to identify the control switches for these hibernator traits.' During hibernation, mammals enter a state of torpor, or physiological dormancy. This allows them to survive months without food and water, but at great cost to their health. Their muscles deteriorate due to lack of nutrition and movement, Christopher Gregg, a professor of neurobiology at U of U and senior author on both studies, told Gizmodo. Proteins associated with Alzheimer's disease build up in their brains, and upon awakening, the sudden reperfusion of blood can cause further neurological damage, he explained. What's more, they become insulin resistant due to the amount of fat they gain to sustain them during months of starvation. Hibernating mammals have evolved remarkable adaptations to reverse this extensive physiological damage. The genes that underlie these adaptations are likely also present in humans and other non-hibernators, Gregg explained. The fact that hibernation has evolved independently in multiple animal species suggests that its basic genetic ingredients are present across the mammalian genome. Therefore, non-hibernators may still carry them. 'We mostly all have the same genes across species,' Gregg said. 'The big change is in the 98% of the genome that does not encode for genes.' Non-coding DNA is largely responsible for gene regulation. In hibernators, specific regions of non-coding DNA act as 'master switches' for controlling functional gene responses to starvation and refeeding, he explained. Finding these master switches in the mammalian genome is like searching for needles in a DNA haystack. To accomplish this, the researchers made whole-genome comparisons across mammals to identify conserved DNA regions that are stable in most species but show accelerated change in hibernators. These hibernator-accelerated regions are regulators that turn genes on in specific cells at specific times, Elliott Ferris, a data analyst in Gregg's lab at U of U and first author of one of the studies, told Gizmodo. To understand the biological processes that may be linked to these hibernator-accelerated regions, the researchers identified genes that get turned up or turned down during fasting in mice. Hibernation is an adaptation to survive food scarcity, so fasting triggers similar metabolic changes. This led them to 'hub genes' that act as master regulators for fasting-induced changes to gene activity. 'The really surprising discovery that was very exciting was that the hibernation-linked elements are disproportionately affecting those key hub genes,' Gregg explained. 'The implication is that hibernators changed the regulation and activity of these core hub genes to have big downstream effects on the whole program for responding to food scarcity and food deprivation. That's important as we think about translating this knowledge into the real world.' Gregg is co-founder of Primordial AI, a Utah-based biotech startup that leverages AI to uncover master regulator gene drug targets. Through this company, he aims to develop drugs that mimic the genetic advantages hibernators have, such as boosting neuroprotection in Alzheimer's patients or reversing insulin resistance in type 2 diabetics. 'Those hub genes are the ones that we think are a really good starting point to design medicines to affect those genes,' Gregg said.


Newsweek
a day ago
- Newsweek
Hidden Human 'Superpowers' Could Help Fight Diabetes
Based on facts, either observed and verified firsthand by the reporter, or reported and verified from knowledgeable sources. Newsweek AI is in beta. Translations may contain inaccuracies—please refer to the original content. Hidden human 'superpowers' could one day help develop new treatments to reverse diabetes and neurodegeneration. This the conclusion of research from University of Utah that suggests hibernating animals' superpowers could lie dormant within our own DNA and could potentially be unlocked to improve our health. "We were fascinated by one big mystery: how do hibernating mammals completely rewire their metabolism—and then reverse it—without damaging their brains or bodies?" Chris Gregg, paper author and U of U Health neurobiology professor, told Newsweek. "They gain huge amounts of fat, become insulin resistant, shut down metabolism and body temperature for weeks or months... and then bounce back without the chronic diseases that plague humans under similar conditions. "That led us to a key idea: maybe the answers aren't in protein-coding genes, which are mostly shared across mammals, but in the noncoding regulatory elements—the parts of the genome that act like switches and dimmers for gene activity." The dangerous health changes hibernating animals can recover from are similar to those seen in type 2 diabetes, Alzheimer's disease, and stroke. It appears humans have the genetic framework to do this, too, if we can figure out how to bypass some of our metabolic switches. A medical doctor using advanced DNA technology, looking at computer screens. A medical doctor using advanced DNA technology, looking at computer screens. AndreyPopov/Getty Images The researchers discovered that a gene cluster called the 'fat mass and obesity (FTO) locus' plays an important role in hibernators' abilities—and humans have these genes too. "What's striking about this region is that it is the strongest genetic risk factor for human obesity," Gregg said in a statement. But hibernators have been observed to be able to use genes in the FTO locus in new ways to their advantage. The team identified hibernator-specific DNA regions near the FTO locus that regulate the activity of neighboring genes, turning them up or down. The research team speculate that this process, including those in or near the FTO locus, allows hibernators to gain weight before settling in for winter. During hibernation, they slowly use these fat reserves for energy. The hibernator-specific regulatory regions outside of the FTO locus seem particularly crucial for tweaking metabolism. When the researchers mutated the hibernator-specific regions in mice, they saw changes in their weight and metabolism. Some mutations sped up or slowed down weight gain under specific dietary conditions, while others affected the ability to recover body temperature after a hibernation-like state or tuned overall metabolic rate up or down, according to the study. Instead of being genes themselves, the hibernator-specific DNA regions the researchers identified were in fact DNA sequences that contact nearby genes and turn their expression up or down, "fine-tuning" them. A hedgehog hibernating in a natural woodland habitat. A hedgehog hibernating in a natural woodland habitat. Callingcurlew23/Getty images "Our research reveals that hidden 'superpowers' from hibernating mammals may be linked to specific genetic elements that exist in human DNA—in the form of conserved, noncoding regulatory elements that control metabolism, brain health and resilience to stress," Gregg explained. "By comparing the genomes of hibernators and non-hibernators, we identified thousands of DNA elements that hibernators have lost or rewired—especially those regulating how the brain responds to fasting and recovery after fasting. When we deleted some of these elements in mice, they changed body weight, energy use, thermoregulation, and even age-related memory behaviors. "One big idea from the study for future testing is that humans have a stable internal body temperature and limited range for metabolic changes and physiological flexibility. The elements we uncovered may act as 'genomic brakes' that limit the range of biological responses to some stressors and hibernators inactivated these DNA elements to evolved expanded capabilities for biological flexibility and resilience." Understanding hibernators' metabolic flexibility could lead to better treatments for human metabolic disorders. However finding the genetic regions that may enable hibernation is no easy feat, with endless DNA to sift through. "What this means is that humans may have the capacity for some aspects of these adaptations, but we would need to unlock them," said Gregg. He explained that if we can understand how to reactivate or mimic these genetic programs, it could lead to new treatments for type 2 diabetes and obesity "by improving metabolic flexibility", Alzheimer's and other neurodegenerative diseases "via the activation of neuroprotective and regenerative gene programs" and stroke and injury recovery "by tapping into natural repair and neuroprotection". It could also potentially help with healthy aging and longevity. A medical device with a hand pointing to a blood sugar meter for diabetes. A medical device with a hand pointing to a blood sugar meter for diabetes. MarcelaTo narrow down the regions involved, the researchers used multiple independent whole-genome technologies to ask which regions might be relevant for hibernation, before looking for overlap between the results from each technique. For example, they looked for sequences of DNA that most mammals share but had recently changed rapidly in hibernators. They also found the genes that act as central coordinators or "hubs" of these fasting-induced changes to gene activity. They discovered that many of the DNA regions that had recently changed also appeared to interact with these central hubs. Hence, the researchers expect that the evolution of hibernation requires specific changes to the controls of the hub genes—helping to narrow down further the DNA elements that are worth further investigation. Crucially, most of the hibernator-associated changes in the genome appeared to "break" the function of specific pieces of DNA, rather than make new functions. This suggests hibernators may have lost constraints that would otherwise prevent extreme flexibility in the ability to control metabolism. "In essence, we're learning to unlock a dormant resilience code in the human genome, using lessons from species that have mastered the art of survival under extreme conditions. It is a big idea that needs much more work to be tested," said Gregg. He said their next areas of focus include testing their "genomic brakes" hypothesis further and exploring the hibernation hub genes and potentially developing them as novel drug targets that enable them to develop medicines that promote metabolic health and neuroprotection. They are also studying hibernation mechanisms in cancer cell evolution and treatment resistance. Do you have a tip on a health story that Newsweek should be covering? Do you have a question about diabetes? Let us know via health@ Reference Ferris, E., Gonzalez Murcia, J. D., Rodriguez, A. C., Steinwand, S., Stacher Hörndli, C., Traenkner, D., Maldonado-Catala, P. J., & Gregg, C. (2025). Genomic convergence in hibernating mammals elucidates the genetics of metabolic regulation in the hypothalamus. Science, 381(6663), 494–500. Steinwand, S., Stacher Hörndli, C., Ferris, E., Emery, J., Gonzalez Murcia, J. D., Rodriguez, A. C., Spotswood, R. J., Chaix, A., Thomas, A., Davey, C., & Gregg, C. (2025). Conserved noncoding cis elements associated with hibernation modulate metabolic and behavioral adaptations in mice. Science, 381(6663), 501–507.


New York Post
6 days ago
- New York Post
New tool can remove nearly all of a cancer-causing ‘forever chemical' from water — in just 5 minutes
Your tap water's dirty little secret might have just met its match. Scientists have engineered a high-tech filter that strips toxic 'forever chemicals' from drinking water in a matter of minutes. Better yet, the new tool glows on contact with contamination, serving as both a purifier and an real-time monitoring system. 4 Research suggests the majority of Americans have 'forever chemicals' in their drinking water. sebra – Forever chemicals — officially known as per- and poly-fluoroalkyl substances (PFAS) — are tiny, man-made compounds that break down very slowly and accumulate in people, animals and the environment over time. They've been used since the 1940s in everything from non-stick cookware and grease-resistant food packaging to waterproof fabrics and personal care products. But concerns are mounting over the potential health effects of PFAS, with some experts likening them to 'slow poison.' One of the most widely used forever chemicals, perfluorooctanoic acid (PFOA), has been linked to higher risks of certain cancers, liver damage, immune system issues, high cholesterol and even developmental delays in fetuses and children. It has been detected across a range of environments and products — from soil and rainfall to seafood, human blood and drinking water. 4 While PFOA was previously used in the manufacturing of non-stock cookware, it has largely been phased out due to concerns about potential health and environmental risks. Dina – In fact, a 2020 study estimated that more than 200 million Americans are drinking water contaminated with PFOA or PFOS, another notorious forever chemical formally known as perfluorooctane sulfonate. Now, researchers at the University of Utah may have found a way to help reduce that exposure. The scientists recently developed a lab-engineered, crystalline substance known as a metal-organic framework (MOF) that functions like a molecular sieve. When water flows through the material, it snags and traps PFOA molecules, effectively removing the forever chemical. 4 The work builds on past research from the team that created a porous material that fluoresces in the presence of PFAS. THE UNIVERSITY OF UTAH In lab tests, the MOF cleared out more than 99% of the toxic substance in just five minutes. 'This sort of rapid treatment is crucial for real-world applications,' the study authors wrote in their report. The material also worked in the presence of other PFAS compounds, salts, and natural organic matter found in drinking water supplies, suggesting it could perform well in a variety of real-world environmental conditions. Better yet, the MOF can be reused, with researchers finding it retained 93% of its adsorption efficiency after five wash cycles. That's a big deal, since current methods for removing PFAS are often slow, costly, inefficient and don't hold up well after repeated use. 4 PFAS in drinking water are estimated to contribute to more than 6,800 cancer cases each year. WESTOCK – An added bonus: when PFOA molecules bind to the material, it glows fluorescent — providing instant, on-the-spot confirmation of contamination. 'This MOF represents a major leap forward for PFAS remediation,' Rana Dalapati, the study's lead author, said in a statement. 'Its ability to both selectively capture and sensitively detect PFOA in real time makes it a versatile and practical solution for water treatment and environmental monitoring,' she added. Looking ahead, the team believes this material could one day be adapted to capture other PFAS chemicals, not just PFOA. Wondering if you should be concerned about forever chemicals in your drinking water? The Environmental Working Group's Tap Water Database is a good place to start — just enter your ZIP code to see what's in your local supply. While the newly developed MOF may one day offer a powerful solution, it will likely be a while before it's available for public use. In the meantime, if you live in an area with known PFAS contamination, consider getting a water filter. Look for one certified by the National Sanitation Foundation to reduce levels of PFOA and PFOS to help limit your exposure.