
Mysterious Antimatter Physics Discovered at the Large Hadron Collider
The world around us is made of matter—the stars, planets, people and things that populate our cosmos are composed of atoms that contain only matter, and no antimatter. But it didn't have to be this way. Our best theories suggest that when the universe was born it had equal amounts of matter and antimatter, and when the two made contact, they annihilated one another. For some reason, a small excess of matter survived and went on to create the physical world. Why? No one knows.
So physicists have been on the hunt for any sign of difference between matter and antimatter, known in the field as a violation of 'charge conjugation–parity symmetry,' or CP violation, that could explain why some matter escaped destruction in the early universe.
On supporting science journalism
If you're enjoying this article, consider supporting our award-winning journalism by subscribing. By purchasing a subscription you are helping to ensure the future of impactful stories about the discoveries and ideas shaping our world today.
Today physicists at the Large Hadron Collider (LHC)'s LHCb experiment published a paper in the journal Nature announcing that they've measured CP violation for the first time in baryons —the class of particles that includes the protons and neutrons inside atoms. Baryons are all built from triplets of even smaller particles called quarks. Previous experiments dating back to 1964 had seen CP violation in meson particles, which unlike baryons are made of a quark-antiquark pair. In the new experiment, scientists observed that baryons made of an up quark, a down quark and one of their more exotic cousins called a beauty quark decay more often than baryons made of the antimatter versions of those same three quarks.
'This is a milestone in the search for CP violation,' says Xueting Yang of Peking University, a member of the LHCb team that analyzed the data behind the measurement. 'Since baryons are the building blocks of the everyday things around us, the first observation of CP violation in baryons opens a new window for us to search for hints of new physics.'
The LHCb experiment is the only machine in the world that can summon sufficient energies to make baryons containing beauty quarks. It does this by accelerating protons to nearly the speed of light, then smashing them together in about 200 million collisions every second. As the protons dissolve, the energy of the crash springs new particles into being.
'It is an amazing measurement,' says theoretical physicist Edward Witten of the Institute for Advanced Study, who was not involved in the experiment. "Baryons containing b [beauty] quarks are relatively hard to produce, and CP violation is very delicate and hard to study.'
The 69-foot-long, 6,000-ton LHCb experiment can track all the particles created during the collisions and the many different ways they can break down into smaller particles. 'The detector is like a gigantic four-dimensional camera that is able to record the passage of all the particles through it,' says LHCb spokesperson and study co-author Vincenzo Vagnoni of the Italian National Institute of Nuclear Physics (INFN). 'With all this information, we can reconstruct precisely what happened in the initial collision and everything that came out and then decayed.'
The matter-antimatter difference scientists observed in this case is relatively small, and it fits within predictions of the Standard Model of particle physics—the reigning theory of the subatomic realm. This puny amount of CP violation, however, cannot account for the profound asymmetry between matter and antimatter we see throughout space.
'The measurement itself is a great achievement, but the result, to me, is not surprising,' says Jessica Turner, a theoretical physicist at Durham University in England, who was not involved in the research. 'The observed CP violation seems to be in line with what has been measured before in the quark sector, and we know that is not enough to produce the observed baryon asymmetry.'
To understand how matter got the upper hand in the early universe, physicists must find new ways that matter and antimatter diverge, most likely via particles that have yet to be seen. 'There should be a new class of particles that were present in the early universe, which exhibit a much larger amount of this behavior,' Vagnoni says. 'We are trying to find little discrepancies between what we observe and what is predicted by the Standard Model. If we find a discrepancy, then we can pinpoint what is wrong.'
The researchers hope to discover more cracks in the Standard Model as the experiment keeps running. Eventually LHCb should collect about 30 times more data than was used for this analysis, which will allow physicists to search for CP violation in particle decays that are even rarer than the one observed here. So stay tuned for an answer to why anything exists at all.

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles
Yahoo
5 hours ago
- Yahoo
Alzheimer's researchers say targeting brain sugar could help protect against dementia
The brain has a hidden "sugar code" that could lead to better treatments for neurological diseases like Alzheimer's, according to new research. A study recently published in the journal Nature Metabolism found that breaking down glycogen (stored glucose) in the brain could reduce the buildup of toxic proteins linked to the common dementia. This was one of the first studies to show that glycogen may actively influence brain health and disease, according to lead author Dr. Pankaj Kapahi, a professor at the Buck Institute for Research on Aging in California. Two Cancer Drugs Show Promise In Reversing Alzheimer's Devastating Effects "The study began with fruit flies (Drosophila) that were genetically modified to mimic tauopathy, a condition where a protein called tau builds up in the brain, similar to what happens in Alzheimer's disease," Kapahi told Fox News Digital. The flies used in the study were found to have brain damage and shortened lifespans, according to the researcher. Read On The Fox News App To ensure that the results could translate to humans, the research team also studied nerve cells made in the lab from human patient cells carrying tau mutations, as well as postmortem brain samples from people who had Alzheimer's or related conditions, according to a press release. In both the fly and human models, the researchers found increased levels of glycogen (stored glucose) in the brain, as well as signs that glycogen breakdown was impaired, Kapahi told Fox News Digital. This was a surprising discovery, as researchers previously thought glycogen was mainly stored in the muscles and liver. Alzheimer's Risk Could Rise With Specific Sleep Pattern, Experts Warn They also found that the excess glycogen contributed to disease. The tau proteins in the scientists' models interacted with glycogen, blocking it from breaking down, and the nerves lost their ability to ward off cell damage. However, the researchers found they could decrease damage to the fruit flies and human nerves by boosting an enzyme called glycogen phosphorylase (GlyP), which breaks down glycogen. The nerve cells used the glycogen to fight against cell damage, suggesting that the enzymes responsible for breaking down sugar could be promising targets for future therapies. The researchers were also curious whether a restricted diet could improve the flies' brain health. When they reduced the amount of protein in the insects' diet, the flies indeed lived longer and their brain health improved. "We then discovered this improvement was linked to an increase in the breakdown of glycogen," Kapahi said. This led to the study's main finding — that breaking down glycogen in neurons can protect the brain from damage caused by tau buildup. Eating These Common Foods Could Reduce Alzheimer's Risk, Experts Say The scientists also developed a drug using a special molecule called 8-Br-cAMP that replicated similar effects of dietary restriction, the press release noted. The authors clarified that they are not recommending low-protein diets just yet — but said this research could pave the way for dietary or drug-based strategies to help slow Alzheimer's and related diseases. Dr. Michael Okun, a Florida neurologist and medical advisor to the Parkinson's Foundation, was not involved in the study but commented on the significance of the findings. "Instead of funneling sugar into energy-burning processes, the broken-down glycogen seemed to be routed into an antioxidant-producing pathway," Okun, who is also author of "The Parkinson's Plan," told Fox News Digital. "The next step in the process may be mopping up potentially damaging free radicals roaming our brains," he said. Okun also confirmed that dietary restrictions activated a protective brain pathway and boosted the breakdown of brain sugar. "It ultimately reduced damage from the Alzheimer's-related tau protein," he said. The study — which was supported by the National Institutes of Health, as well as the American Federation of Aging Research and other sources — did have some limitations, the experts acknowledged. Click Here To Sign Up For Our Health Newsletter The researchers did not clarify whether breaking down brain glycogen could stop human brain cells from dying, Okun noted. "We don't yet know why glycogen builds up in disease, or whether it's a cause or a result of tau pathology — though our data suggest it may amplify disease progression," Kapahi added. The research was also conducted only on fly and human cell models and has not yet been tested on living humans. "We still don't know if targeting glycogen breakdown will work in human patients — and most importantly, whether it will be a safe approach," Okun said. Alzheimer's disease, the most common form of dementia in the U.S., affects more than seven million people in the U.S., according to the Alzheimer's Association. For more Health articles, visit The neurological disorder affects memory, thinking and behavior. There is no cure for the disease, but some medications can temporarily slow progression and improve quality of article source: Alzheimer's researchers say targeting brain sugar could help protect against dementia Solve the daily Crossword


CNET
14 hours ago
- CNET
This Nature App Uses AI to Turn Anyone Into a Wildlife Expert. Here's How to Get Started
When I recently moved, one of the building's standout features was the plethora of different plant species in front of its windows. But not like standard landscaping -- more like drier plants, seemingly native to the area. Curious and fairly incompetent in plant sciences, I looked for help in being able to categorize and understand more about it. That's how I found Seek by iNaturalist, a free app where you take photos of plants, animals and fungi. Artificial intelligence suggests its type -- with human confirmation -- while your input and contributions elevate scientific research. What is Seek and how does it use AI? Founded in 2008 as a master's project from UC Berkeley students, iNaturalist later became a joint initiative between the California Academy of Sciences and the National Geographic Society. It strives to connect people to nature and conservation through crowdsourced data. Today, it contains more than 250 million observations of more than a half-million species. (And I just added seven more in roughly 5 minutes.) Seek by iNaturalist is 100% free thanks to donors who contributed to the platform. It operates as a nonprofit organization that makes all collected data -- with the option to turn it off -- openly accessible to scientists and conservationists worldwide. iNaturalist uses computer vision AI to analyze uploaded photos against a model trained on millions of previous observations. AI is also used on the back end -- it optimizes species recognition accuracy, to display the most relevant photos alongside suggestions and pattern recognition improvements. This helps you distinguish between similar-looking species at home or across your local ecosystems. According to the iNaturalist team, "When you use the AI camera feature on any wild living thing, iNaturalist suggests an identification for what you might be seeing -- whether plant, insect, fungus or other organism." They shared that from there, that photo becomes a useful piece of data for science and conservation. But ultimately, a community of naturalists confirm or correct those AI suggestions. Together, AI and human intelligence help newcomers contribute to biodiversity documentation from their first upload. How to use iNaturalist to spot and categorize nature 1. First search for Seek in your app store of choice and create an account by signing up with email or a social media authenticator. You'll also need to allow camera and location permissions to be able to use the AI's computer vision and accurately detect species. 2. It's best to play around a bit before starting scans. Open the app and tap the camera button -- it's designed to recognize any plant, animal and fungi. I focused solely on plants, as that's what I had readily available. Snap a couple photos or hold the camera button down to snap continuously. Using iNaturalist's AI camera to identify a plant species. Screenshot by CNET As you're testing the camera out, note background images, angles and even lighting, which all play a part in the AI tool's ability to accurately recognize what you've captured. (I had to redo a couple because an attempt to take a birds-eye shot ended up with pictures of my feet.) 3. Next, the AI computer vision will analyze your photo and suggest possible species. This takes no longer than 30 seconds. Here you can also record GPS coordinates and you have the option to add details about behavior, size and other aspects of whatever it is you observed and captured. Beyond this, you can also toggle between privacy options, and select whether it is captive, like a plant, or wild, like natural growth. 4. AI also has suggestions for your species identification, which show up as percentages for accuracy. You can also post your observation -- if geoprivacy is open -- to the community for review by users and experts, like scientists. Once the community confirms your species, your observation becomes scientific data. The point here is that you are part of the collection process for scientists to learn more about species in the world. With your input and the community's approval, you're directly involved in this process. Think of yourself as a bridge between reality and what scientists are constantly trying to understand and improve. The iNaturalist team provided tips for getting the best results, alongside checking out their company guides, or downloading the Seek user guide: treat AI suggestions as a starting point, and engage with other humans; explore information on iNaturalist's database; and be aware that there are more advanced camera suggestions in the iPhone app over the Android app. Should you use iNaturalist? The app is designed like Instagram for nature lovers, with a volunteer process that positions itself as additional help for scientists' discoveries. In my opinion, technology that helps connect people to nature and advances scientific understanding is inherently valuable for our planet and future generations. Some of the privacy options available on the iNaturalist Seek app when you upload your observations. iNaturalist / Screenshot by CNET But even though Seek operates globally, with 3.7 million users, the identification accuracy depends heavily on having active expert communities in your region. This means rural or less-populated areas might get slower species confirmations. Additionally, the topic of AI ethics comes to mind. I asked the iNaturalist team about this. They responded that the location is important for information when correctly identifying species, especially wild ones. Yet they also noted that anyone using iNaturalist can "choose to obscure the precise locations of observations to protect their privacy" and that you can use the AI identification feature without creating an account or posting publicly. The iNaturalist team also regularly updates the Privacy Policy in support of the well-being of the community. The main goal is to create an open biodiversity dataset that can be used to protect nature around the world. Overall, Seek by iNaturalist is a great way to educate yourself and contribute to multiple versions of ecosystems in the process while learning and sharing your findings along the way -- or, as the iNaturalist team shared, a "virtuous cycle of collective effort and improvement, built on community trust and benefit in service of helping people connect with nature and biodiversity worldwide."


The Hill
3 days ago
- The Hill
Flu, COVID can reawaken dormant breast cancer cells: Study
Respiratory infections like COVID-19 and the flu can activate dormant cancer cells in breast cancer patients who are in remission, new research finds. The study, published in Nature, found that common viruses can reawaken small numbers of dormant breast cancer cells in the lungs. Researchers began investigating the link after the team noticed that U.K. patients who were in remission from breast cancer and tested positive for COVID later showed a two-fold increase in cancer-related deaths. They also analyzed a U.S. database that included nearly 37,000 patients and found that COVID infection was associated with a more than 40 percent increased risk of metastatic breast cancer in the lungs. Studies on mice found that influenza and COVID infections triggered dormant breast cancer cells after just days of infection. Within two weeks, researchers observed 'massive expansion' of the cancer cells into metastatic lesions by more than 100 times. Scientists have suspected that common viruses like Epstein-Barr can trigger some cancers. Human papillomavirus (HPV) is already documented to trigger cervical cancer. When it comes to breast cancer, however, research on human cells was limited, and it's not entirely known how the virus triggers the disease to spread. The findings suggest the body's immune response plays a role. After breast cancer goes into remission, a tiny number of cells remain dormant in lung, bone and liver tissue. Sometimes, inflammation can wake up the cells. In the mouse experiments, both influenza A and coronavirus only reawakened dormant cells if they triggered an inflammatory cytokine response. More research is needed to see if vaccination makes a difference when it comes to the possibility of reawakening dormant cells.