
Belly fat ‘may cause burning and painful skin condition' suffered by Kim Kardashian – how to shift the pounds
Psoriasis, which affects one in 50 people - including reality TV star Kim Kardashian - causes scaly patches of skin usually around the scalp, knees and elbows.
5
5
The 44-year-old has previously opened up about her struggle with the condition she described as 'painful'.
Model Cara Delevingne and singer Cyndi Lauper are also sufferers.
Researchers at King's College London analysed data from over 330,000 Brits, including more than 9,000 people with psoriasis.
They found that abdominal fat was strongly linked to someone's likelihood of having the condition, particularly for women.
Study author Dr Ravi Ramessur said: 'Our research shows that where fat is stored in the body matters when it comes to psoriasis risk.
'Central fat, especially around the waist, seems to play a key role.
'This has important implications for how we identify individuals who may be more likely to develop psoriasis or experience more severe disease, and how we approach prevention and treatment strategies.'
Psoriasis typically begins in someone's 20s or 50s.
It's not known what causes the incurable, and therefore chronic, condition.
5
5
5
It is thought that a problem with the immune system causes the skin cells to replace faster than they should. It is therefore sometimes referred to as an autoimmune disorder.
Dr Ramessur says: "The observed links between central body fat and psoriasis suggest that there may be underlying biological mechanisms contributing to the disease that are not yet fully understood and which warrant further investigation."
Khloe Kardashian leaks 'creepy' video of sister Kim's legs on Snapchat as fans say 'this is so strange!'
In light of the new findings, experts said Ozempic-style fat jabs, such as Wegovy and Mounjaro, also known as GLP-1 receptor agonists, may help people with psoriasis.
Dr Joel Gelfand, Department of Dermatology and Center for Clinical Sciences in Dermatology, University of Pennsylvania Perelman School of Medicine, wrote in an accompanying editorial: 'The strong relationship between psoriasis and obesity, and the emerging promise of GLP-1 RAs, is a call to action for clinical trials.'
Dr Catherine Smith, senior author, said: 'As rates of obesity continue to rise globally, understanding how different patterns of body fat influence chronic inflammatory conditions such as psoriasis is important."
How to get rid of belly fat
Most of us have tried to shift belly fat at some point - it can be stubborn and takes some hard work.
Personal trainer Lucy Gornall previously shared with The Sun her non-negotiables if your goal is to lose the extra chub around your middle.
These include:
1. Fill your plate with as many coloured fruits and vegetables as you can.
"These are the fruits and vegetables which pack in nutrients and fibre, but contain very few calories," Lucy says.
Fibre fills you up and therefore prevents overeating.
2. Don't overdo the cardio.
"Going hard on runs, cycles and other types of cardio, is NOT the best way to burn belly fat," says Lucy.
"It will leave you tired (you'll be turning to high sugar snacks for energy), it will leave you sore, and it will leave you very hungry."
3. Instead, lift weights.
"More muscle means more calories burnt at rest, meaning you burn off the food you eat easier than if you had less muscle," says Lucy.
"Aim to include three 30 to 45 minute resistance-based training sessions each week, ensuring that the weight is challenging and your form is correct."
4. Eat protein
Protein is the most filling food you can eat (over carbohydrates and fats) and helps with muscle recovery from all that weight training you'll be doing.
"Look to include a source of protein in every meal and you won't go too far wrong," says Lucy, noting chicken breast and lean turkey mince are good sources.
5. Move more generally.
Increase your NEAT (non-exercise activity thermogenesis) by doing things like taking the stairs over the lift and walking home from an earlier train or tube station.
What is psoriasis?
Roughly two per cent of the population - both men and women - are affected by psoriasis.
Prominent figures that deal with the skin problem are model Cara Delevingne and singer Cyndi Lauper.
TV personality Kim Kardashian has also described psoriasis as her 'big flaw', always 'hoping for a cure'.
Sadly, there is no cure for psoriasis.
Patients have to learn to deal with the condition by finding a treatment that works for them.
But some have also found that modifying their diet helps.
Treatments
The NHS says: 'A wide range of treatments are available for psoriasis, but identifying the most effective one can be difficult.'
Treatments fall into three categories.
First, topical creams and ointments applied to the skin are usually the first option to help with mild psoriasis.
If the condition has spread to the scalp, there are also shampoos and ointments that can help.
Examples include:
Emollients to keep the skin moist and reduce water loss
Steroid creams - which are only prescribable - to calm the skin
Vitamin D analogues to slow the production of skin cells and reduce inflammation
Calcineurin inhibitors to dampen inflammation and the immune system, sometimes in sensitive areas
Coal tar to reduce scales and itchiness
Dithranol to suppress the production of skin cells
Hashtags

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles


Reuters
a minute ago
- Reuters
UK scientists find genes linked with chronic fatigue syndrome
Aug 6 (Reuters) - Researchers at the University of Edinburgh said on Wednesday they have discovered differences in the DNA of people with chronic fatigue syndrome that should help dispel the notion that the debilitating condition is psychological or driven by laziness. Their study found eight areas of genetic code that are different in people with myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) than in healthy volunteers. The finding provides 'the first robust evidence that genes contribute to a person's chance of developing the disease,' the researchers said in a statement. The key features of the condition include worsening of fatigue, pain and brain fog after even minor physical or mental activity. Very little has been known about the causes of ME/CFS, and there is no diagnostic test or cure. The condition is believed to affect around 67 million people worldwide, the researchers said. The DecodeME study analyzed DNA samples from 15,579 people who reported having chronic fatigue on a questionnaire and 259,909 people without it, all of European descent. Gene variants that were more common in people reporting ME/CFS were linked to the immune and nervous systems, according to a report of the study that has not yet been peer-reviewed. At least two of the gene regions relate to how the body responds to infection, which aligns with reports that the symptoms often start after an infectious illness, the researchers said. Another gene region has previously been identified in people with chronic pain, another common symptom of the condition. The findings 'align with decades of patients reporting on their experiences,' researcher Andy Devereux-Cooke said in a statement, adding that they "should prove game changing in the ME/CFS research field.' 'These results will not mean that a test or cure will be developed straight away, but they will lead to a greater understanding," he said. Scientists who were not involved in the study said using volunteers who self-reported chronic fatigue syndrome rather than restricting participation to those with a diagnosis from a medical professional somewhat weakened its conclusions. They called for larger studies to replicate the results. Substantial work will be necessary 'to translate these findings into new treatments," said Dr. Jackie Cliff, who studies infection and immunity in ME/CFS at Brunel University of London. "This will take considerable investment in academia and by industry.'


Times
an hour ago
- Times
Breakthrough genetic study offers treatment hope for ME patients
Myalgic encephalomyelitis (ME) could have genetic roots, researchers have found, in the biggest scientific breakthrough yet into the disease. A team at the University of Edinburgh analysed DNA samples from people with ME, also known as chronic fatigue syndrome (CFS), in a hunt to uncover the biological roots of the illness. About 400,000 people in Britain have the disease, which leads to profound exhaustion and pain, but until now scientists knew 'next to nothing' about what causes it. The new study revealed eight 'signals' in genetic code where people with ME tended to have markedly different gene variants compared to the healthy population. It is the first robust evidence that genes predispose people to the disease and suggests that ME may be caused by processes in the immune and nervous systems.


Daily Mail
an hour ago
- Daily Mail
Scientists FINALLY crack the mystery of chronic fatigue syndrome - major breakthrough reveals cause, sparks new hope for effective treatment
Scientists have uncovered a biological cause of chronic fatigue syndrome in a breakthrough that promises to transform understanding of the debilitating illness. The condition—long dismissed by some as psychological—has now been linked to clear genetic differences found in patients' DNA. Researchers say the findings offer the first robust evidence that inherited genes can influence the risk of developing chronic fatigue syndrome (CFS), also known as myalgic encephalomyelitis (ME). Experts say the discovery gives 'validity and credibility' to patients and helps to dispel the stigma and disbelief that often surrounds the illness. The landmark study by the University of Edinburgh identified eight distinct genetic markers in people with the condition, compared to those without. Professor Chris Ponting, who led the DecodeME project, said: 'This is a wake-up call. The eight genetic signals reveal much about why infection triggers ME/CFS and why pain is a common symptom. 'ME/CFS is a serious illness and we now know that someone's genetics can tip the balance on whether they are diagnosed with it.' Researchers analysed DNA from more than 15,000 people with ME/CFS, part of the world's largest study into the disease. The eight regions of DNA where scientists found differences involve genes linked to the immune and nervous systems. At least two of the signals relate to how the body responds to infection, echoing long-standing patient reports that symptoms often begin after an illness. Previous studies have shown that people who catch Covid are up to seven times more likely to develop ME/CFS, which shares symptoms with Long Covid. There is currently no diagnostic test or cure for the condition, which is thought to affect around 67 million people worldwide. In the UK alone, an estimated 404,000 people are affected—with women more likely to suffer than men, though researchers found no clear reason for this. Well-known sufferers include comedian Miranda Hart, who revealed in her autobiography, which was published last year, the condition left her 'bedbound and without joy'. Symptoms include pain, brain fog and a profound lack of energy. A key feature of ME/CFS is post-exertional malaise—a severe worsening of symptoms after even minor physical or mental activity. Scientists hope the findings will pave the way for better treatments, understanding and recognition of the illness. Sonya Chowdhury, Chief Executive of Action for ME and a DecodeME co–investigator, said: 'These results are groundbreaking. With DecodeME, we have gone from knowing next to nothing about the causes of ME/CFS, to giving researchers clear targets.' She added: 'This really adds validity and credibility for people with ME. We know that many people have experienced comments like ME is not real, or they've been to doctors and been disbelieved or told that it's not a real illness. 'Whilst things have changed and continue to change, that is still the case for some people and we hear that repeatedly as a charity. 'Being able to take this study into the treatment room and say there are genetic causes that play a part in ME is going to be really significant for individuals. It will rebuff that lack of belief and the stigma that exists.'