logo
It's time to take the fear out of healthcare advertising and replace it with joy, hope, and authentic connection

It's time to take the fear out of healthcare advertising and replace it with joy, hope, and authentic connection

Fast Company18-07-2025
There's rarely a week that goes by without me having a conversation with a colleague or client about the ineffectiveness of fear-based advertising. For too long, our industry has relied on tropes of fear, shame, and judgment to motivate patients and providers. A friend of mine calls this 'sadvertising.'
Health advertising's premise has been deceptively simple: 'If people just knew the dangers, they would alter their behaviors.'
We've become masters of euphemism, leveraging scare tactics under the guise of 'creating urgency.' But beneath the marketing -speak lies an uncomfortable truth. We've built a category lexicon of dark messages and imagery that perpetuates a culture of sickness instead of inspiring wellness.
In doing so, we've stifled creativity and conditioned marketers and agencies to approach too many briefs (whether consciously or not) through a fear-based lens that may grab attention in the short term, but rarely leads to lasting change.
The problem is fear isn't working.
It's time we stop creating work that holds up a mirror to what's broken and start imagining creative solutions that inspire a new vision of what's possible.
THE EVIDENCE AGAINST FEAR
Statistically, scientifically, and psychologically, fear hardly ever drives long-term impact.
The Surgeon General's warning first appeared on cigarettes in 1964, yet smoking rates continued climbing for decades. The 'war on drugs' consumed over $1T in fear-based messaging, yet overdose deaths increased, stigma spread, and healthcare systems still buckle under the strain today. Similarly, over 50% of new drug launches fail to meet forecasts, and most wellness products don't live past year one.
There's been no shortage of ways advertising has tried to guilt people into caring for their health.
We've deployed everything from anxiety-inducing warnings ('diabetes could cost you your toes') and catastrophic scenarios ('meningitis can kill in hours') to hair-raising statistics, dark imagery, stern voice-overs, and ominous soundtracks.
In healthcare, where life-and-death consequences feel immediate, fear may initially seem like the most efficient path to behavior change. But time and again, fear backfires, eroding the very trust we need to build.
The opportunity isn't in perfecting new forms of fear—it's in challenging brands to pivot toward joy.
A CREATIVE RENAISSANCE AWAITS
Earlier this month, brands across nearly every industry and continent gathered on the French Riviera to celebrate great storytelling at the Cannes Lions International Festival of Creativity. For health advertisers, it was an important moment to take a step back and ask: Can new forms of creativity help improve and save lives?
Here's where we can begin forging a better path forward.
Entertainment As A Secret Weapon Of Behavior Change
Netflix's Live to 100: Secrets of the Blue Zones doesn't lecture viewers about longevity. It invites them into communities where people naturally live healthier, longer lives through connection and purpose.
AXA's Group Therapy campaign took a similar approach, trading fear for humor and connection. Featuring celebrities like Neil Patrick Harris in a docu-style format, it tackled mental health through empathetic storytelling, not shock value. By spotlighting shared vulnerability, it made mental health feel more relatable and more human.
Similarly, New Zealand's Cannes Grand Prix winning 'Best Place to Have Herpes' campaign. The campaign tackled taboo with humor and heart by launching a crash-course on destigmatization. It invited people to talk about the issue, laugh about it, and most importantly, take it seriously without shame.
Accessibility And Inclusion As Creative Foundation
The most powerful health campaigns meet people where they are, not where we think they should be.
Eli Lilly's recent diabetes campaigns showcase real people living full, empowered lives by emphasizing possibility over complications. Similarly, Aveeno's 'Eczema Equality' initiative celebrates diverse skin by featuring confident families and children, transforming a condition often hidden in shame into a source of authentic representation and acceptance.
Health messaging becomes most effective when building bridges, not putting up barriers. Apple's accessibility-focused campaigns, 'The Greatest' and 'The Relay,' demonstrate how brands can embed inclusivity into their core messaging.
And in a world where nearly 53% of Gen Z identify with some form of neurodiversity and 129 million Americans have pre-existing conditions, the opportunities for inclusive marketing are endless. When brands prioritize accessibility, they don't just reach more people; they create more authentic connections.
Vaseline's Vaseline Verified campaign echoed this approach by also meeting Gen Z on TikTok. By testing trending 'hacks' in real labs and educating through science, it showed how trust and transparency can outshine fear and misinformation.
Conversation Placement: The New Product Placement
We all remember when Reese's Pieces led E.T. home in 1982, launching the modern era of product placement. Since then, we've seen Heineken in James Bond films, Ray-Bans in Top Gun, and Coca-Cola in Stranger Things. Brands invest massive marketing dollars to infiltrate our entertainment with products they want us to buy.
But what if we started placing health conversations into our entertainment instead? My colleague Andre Gray calls this ' conversation placement. '
Instead of inserting sneakers into a TV series, what if we featured an attorney with HIV on Law & Order or a teenager with cystic fibrosis navigating high school drama? We've gotten a glimpse of this potential already. Everything Everywhere All at Once showed us Evelyn Wang living with ADHD, Glee gave us Artie Abrams as a wheelchair user, and The Good Doctor brought us Shaun Murphy, a surgeon with autism and savant syndrome.
'Conversation placement' tackles real health issues with real people and bridges gaps through culture and inclusion.
THE JOY IMPERATIVE
Fear feels reliable because it's easily manufactured. But fear creates lose-lose scenarios by breeding resentment toward the very health systems and behaviors we want people to embrace.
As the dust settles post-Cannes Lions, let's remember that the most meaningful creative work doesn't shock people into change; it invites them in. The path forward isn't about abandoning all urgency or glossing over real health risks. It's about communicating those realities through frameworks of hope, community, and empowerment.
When healthcare advertising embraces joy, laughter, and authentic human connection, we don't just create better campaigns—we create better health outcomes. We build trust instead of eroding it.
So, when in doubt, choose joy. Your audience (and their health) will thank you for it.
Orange background

Try Our AI Features

Explore what Daily8 AI can do for you:

Comments

No comments yet...

Related Articles

The Silent Symptom That Could Be a Sign of Blood Clots
The Silent Symptom That Could Be a Sign of Blood Clots

Yahoo

time36 minutes ago

  • Yahoo

The Silent Symptom That Could Be a Sign of Blood Clots

The Silent Symptom That Could Be a Sign of Blood Clots originally appeared on Parade. Your blood's ability to clot after a cut or injury is an important defense mechanism to keep you from bleeding too much. But blood clots can cause serious health issues like strokes and heart attacks when they happen outside of that. About 900,000 people in the U.S. develop a blood clot each year, according to the Centers for Disease Control and Prevention (CDC). An estimated 60,000 to 100,000 Americans die from blood clot complications annually, making this an important health issue to be aware of. 'Awareness of blood clot symptoms is critically important because early recognition can be life-saving,' says, a pulmonologist at The Ohio State University Wexner Medical Center. 'Recognizing symptoms early allows for prompt medical treatment.' That can help lower the risk of permanent damage to organs or tissues, he points the problem: Not all symptoms of blood clots are obvious, and one in particular can be easily mistaken for other, much less severe health issues. Here's what doctors want you to keep in mind about this symptom, why it can be confused with other things and when to take action. 🩺SIGN UP for tips to stay healthy & fit with the top moves, clean eats, health trends & more delivered right to your inbox twice a week💊 How Do Serious Blood Clots Happen? Before we go over the symptom, it's important to first go over how blood clots can become serious. Blood clots that happen spontaneously usually start in the legs, explains , a vascular surgeon at MemorialCare Orange Coast Medical Center in Fountain Valley, California. 'They aren't life-threatening in themselves, but they occasionally dislodge and go to the lungs,' he says. This is called a pulmonary embolism. Related: With a pulmonary embolism, a blood clot gets stuck in an artery in the lung and blocks blood flow to part of the lung, Dr. Yi explains. There, it can cause permanent damage to the lungs, low oxygen levels in your blood, and damage to other organs in your body (from not getting enough oxygen), he says, adding, 'This can be life-threatening." The Silent Sign of Blood Clots To Know About, According to a Vascular Surgeon Back to that silent symptom: Shortness of breath is common with pulmonary embolisms. 'Shortness of breath is a hallmark symptom of pulmonary embolism because of how the condition affects the lungs and oxygen delivery,' Dr. Parson says. 'The blocked artery prevents blood from reaching parts of the lung, so oxygen can't be absorbed into the bloodstream efficiently, resulting in shortness of breath.' This blockage usually comes on suddenly, so the body doesn't have time to compensate for the lower-than-usual oxygen, he explains. That can lead to sudden and intense shortness of breath. Related: When Shortness of Breath Is a Sign of a Blood Clot Shortness of breath can also be a sign of a slew of other things, including being out of shape, having asthma or just having a cold, making this a tricky thing to pin on a blood clot. But doctors say there are a few key differences between 'regular' shortness of breath and feeling breathless due to a blood clot.'Standard shortness of breath usually comes with exercise or activity,' Dr. Yi says. 'With a blood clot, there is a sudden onset of shortness of breath where you feel like you can't catch your breath.' You may also start breathing faster than usual, he explains. Along with coming on hard and fast, shortness of breath from a blood clot tends to get worse with exertion or taking deep breaths, according to Dr. Other Signs of a Blood Clot and What To Do While sudden shortness of breath alone should raise concerns about a possible pulmonary embolism, there are other blood clot symptoms doctors warn should be on your radar: Fast breathing Chest pain (it usually gets worse when you cough or take a deep breath) A faster-than-usual heart rate Coughing, including coughing up blood Very low blood pressure Feeling lightheaded Fainting It can be tempting to write these signs off if you have one or two that can be explained away as something more minor. But doctors stress the importance of taking these seriously if you or someone around you has them. 'If someone experiences these symptoms, immediate medical attention is critical,' Dr. Parsons says. Up Next:Sources: How Does Blood Clot? National Heart, Lung, and Blood Institute Data and Statistics on Venous Thromboembolism, Centers for Disease Control and Prevention Dr. Christopher Yi, MD, a vascular surgeon at MemorialCare Orange Coast Medical Center in Fountain Valley, CA Pulmonary Embolism. US National Library of Medicine Dr. Jonathan Parsons, MD, a pulmonologist at The Ohio State University Wexner Medical Center The Silent Symptom That Could Be a Sign of Blood Clots first appeared on Parade on Jul 27, 2025 This story was originally reported by Parade on Jul 27, 2025, where it first appeared. Solve the daily Crossword

Millions of people are suffering from brain fog. A new study will find out why
Millions of people are suffering from brain fog. A new study will find out why

Yahoo

timean hour ago

  • Yahoo

Millions of people are suffering from brain fog. A new study will find out why

Millions of people who recover from infections like COVID-19, influenza and glandular fever are affected by long-lasting symptoms. These include chronic fatigue, brain fog, exercise intolerance, dizziness, muscle or joint pain and gut problems. And many of these symptoms worsen after exercise, a phenomenon known as post-exertional malaise. Medically the symptoms are known as myalgic encephalomyelitis or chronic fatigue syndrome (ME/CFS). The World Health Organization classifies this as a post viral fatigue syndrome, and it is recognised by both the WHO and the United States Centers for Disease Control and Prevention as a brain disorder. Experiencing illness long after contracting an infection is not new, as patients have reported these symptoms for decades. But COVID-19 has amplified the problem worldwide. Nearly half of people with ongoing post-COVID symptoms – a condition known as long-COVID – now meet the criteria for ME/CFS. Since the start of the pandemic in 2020, it is estimated that more than 400 million people have developed long-COVID. To date, no widely accepted and testable mechanism has fully explained the biological processes underlying long-COVID and ME/CFS. Our work offers a new perspective that may help close this gap. Our research group studies blood and the cardiovascular system in inflammatory diseases, as well as post-viral conditions. We focus on coagulation, inflammation and endothelial cells. Endothelial cells make up the inner layer of blood vessels and serve many important functions, like regulating blood clotting, blood vessel dilation and constriction, and inflammation. Our latest review aims to explain how ME/CFS and long-COVID start and progress, and how symptoms show up in the body and its systems. By pinpointing and explaining the underlying disease mechanisms, we can pave the way for better clinical tools to diagnose and treat people living with ME/CFS and long-COVID. What is endothelial senescence? In our review, our international team proposes that certain viruses drive endothelial cells into a half-alive, 'zombie-like' state called cellular senescence. Senescent endothelial cells stop dividing, but continue to release molecules that awaken and confuse the immune system. This prompts the blood to form clots and, at the same time, prevent clot breakdown, which could lead to the constriction of blood vessels and limited blood flow. By placing 'zombie' blood-vessel cells at the centre of these post-viral diseases, our hypothesis weaves together microclots, oxygen debt (the extra oxygen your body needs after strenuous exercise to restore balance), brain-fog, dizziness, gut leakiness (a digestive condition where the intestinal lining allows toxins into the bloodstream) and immune dysfunction into a single, testable narrative. From acute viral infection to 'zombie' vessels Viruses like SARS-CoV-2, Epstein–Barr virus, HHV-6, influenza A, and enteroviruses (a group of viruses that cause a number of infectious illnesses which are usually mild) can all infect endothelial cells. They enable a direct attack on the cells that line the inside of blood vessels. Some of these viruses have been shown to trigger endothelial senescence. Multiple studies show that SARS-CoV-2 (the virus which causes COVID-19 disease) has the ability to induce senescence in a variety of cell types, including endothelial cells. Viral proteins from SARS-CoV-2, for example, sabotage DNA-repair pathways and push the host cell towards a senescent state, while senescent cells in turn become even more susceptible to viral entry. This reciprocity helps explain why different pathogens can result in the same chronic illness. Influenza A, too, has shown the ability to drive endothelial cells into a senescent, zombie-like state. What we think is happening We propose that when blood-vessel cells turn into 'zombies', they pump out substances that make blood thicker and prone to forming tiny clots. These clots slow down circulation, so less oxygen reaches muscles and organs. This is one reason people feel drained. During exercise, the problem worsens. Instead of the vessels relaxing to allow adequate bloodflow, they tighten further. This means that muscles are starved of oxygen and patients experience a crash the day after exercise. In the brain, the same faulty cells let blood flow drop and leak, bringing on brain fog and dizziness. In the gut, they weaken the lining, allowing bits of bacteria to slip into the bloodstream and trigger more inflammation. Because blood vessels reach every corner of the body, even scattered patches of these 'zombie' cells found in the blood vessels can create the mix of symptoms seen in long-COVID and ME/CFS. Immune exhaustion locks in the damage Some parts of the immune system kill senescent cells. They are natural-killer cells, macrophages and complement proteins, which are immune molecules capable of tagging and killing pathogens. But long-COVID and ME/CFS frequently have impaired natural-killer cell function, sluggish macrophages and complement dysfunction. Senescent endothelial cells may also send out a chemical signal to repel immune attack. So the 'zombie cells' actively evade the immune system. This creates a self-sustaining loop of vascular and immune dysfunction, where senescent endothelial cells persist. In a healthy person with an optimally functioning immune system, these senescent endothelial cells will normally be cleared. But there is significant immune dysfunction in ME/CFS and long-COVID, and this may enable the 'zombie cells' to survive and the disease to progress. Where the research goes next There is a registered clinical trial in the US that is investigating senescence in long-COVID. Our consortium is testing new ways to spot signs of ageing in the cells that line our blood vessels. First, we expose healthy endothelial cells in the lab to blood from patients to see whether it pushes the cells into a senescent, or 'zombie,' state. At the same time, we are trialling non‑invasive imaging and fluorescent probes that could one day reveal these ageing cells inside the body. In selected cases, tissue biopsies may later confirm what the scans show. Together, these approaches aim to pinpoint how substances circulating in the blood drive cellular ageing and how that, in turn, fuels disease. Our aim is simple: find these ageing endothelial cells in real patients. Pinpointing them will inform the next round of clinical trials and open the door to therapies that target senescent cells directly, offering a route to healthier blood vessels and, ultimately, lighter disease loads. Burtram C. Fielding is Dean Faculty of Sciences and Professor in the Department of Microbiology, Stellenbosch University This article is republished from The Conversation under a Creative Commons license. Read the original article.

Human Babies Aren't Supposed to Have 3 Parents—but Now They Can
Human Babies Aren't Supposed to Have 3 Parents—but Now They Can

Yahoo

time2 hours ago

  • Yahoo

Human Babies Aren't Supposed to Have 3 Parents—but Now They Can

"Hearst Magazines and Yahoo may earn commission or revenue on some items through these links." Here's what you'll learn when you read this story: The first babies with three biological parents were born out of a new technique to prevent mitochondrial disease. The nucleus of an egg fertilized in vitro was transferred into a donor egg without a nucleus, but with viable mitochondria. Eight healthy babies, including a set of twins, were born with low to undetectable levels of mitochondrial mutations. The only creatures known to conceive offspring from more than two parents are salamanders. Females from the genus Ambystoma (which are notoriously promiscuous) mate with up to three different males, and that DNA is then incorporated into what is known as a triploid genome in their offspring. Now a version of this has become possible in humans. It seems limb regeneration isn't the only way medical intervention can put humans on salamanders' level. Being born with three genomes is not a phenomenon that occurs naturally in Homo sapiens, but in an attempt to prevent certain genetic conditions caused by mutations in the mitochondria, scientists have found a way. Mitochondrial DNA or mtDNA is exclusively passed down from the maternal side. Dysfunction in the mitochondria can lead to metabolic diseases characterized by symptoms such as seizures, developmental delays, blindness, and loss of muscular function. Some can even be fatal. Mitochondrial diseases occur in about 1 in every 5,000 people. They were previously only preventable by using a donor egg or foregoing the conception of biological children altogether. This is why pediatric neurologist Bobby McFarland, of Newcastle University in the UK, led an experimental study that would reduce and potentially eliminate the risk of mitochondrial disease with a new method of in vitro fertilization. McFarland and his research team wanted see if removing the nucleus of an egg and placing it in a donor egg with viable mitochondria would result in healthy offspring. 'We found that pronuclear transfer, a form of mitochondrial donation, was effective in reducing the level of pathogenic mtDNA variant to substantially below the threshold for clinical disease in the offspring of women with homoplasmic (or high heteroplasmic) levels,' he said in a study recently published in the New England Journal of Medicine. When mitochondria are homoplasmic, all copies produced by cell division have mutations. Mutation levels vary in heteroplasmic mitochondria. Preimplantation genetic testing (PGT) can screen for these abonormalities, and women with homoplasmy or high levels of heteroplasmy can benefit from what is now known as pronuclear transfer. This involves eggs from both the mother and donor being fertilized with the father's sperm in vitro. Nuclei are then removed from both eggs after ten hours. Since the nucleus carries most genetic material and has no connection to mitochondrial disease, the mother's nucleus is implanted into the donor egg to take advantage of its mitochondria. While there is a chance that a few of the mother's mitochondria may end up in the embryo, it is unlikely to cause a debilitating disease. Levels of defective mitochondria in offspring conceived via pronuclear transfer were low enough to escape that fate. Eight pregnancies (including a set of twins) resulted from the experiment, and while there were a few minor health problems in the newborns, these were either treatable or corrected themselves. Not only were levels of heteroplasty low for the babies, but undetectable in five of them. Developmental progress also turned out to be normal. Though one baby had a form of infant epilepsy, and another had heart arrhythmia and hyperlipidemia, or high levels of fats and lipids in the blood, both of these conditions were treated and resolved. Whether the hyperlipidemia was even caused by mtDNA is uncertain, especially because the mother also had severe hyperlipidemia during her pregnancy. Though there was a chance that any of the mothers with pathogenic mtDNA had a higher risk of complications during pregnancy, which could possibly cause their children to have health issues, there is no proof for now. 'We are assessing, over the long term, the health and extent of heteroplasmy (if detectable) of the offspring,' McFarland and his team said. 'Indeed, the role of mitochondrial donation as a choice for women with a heritable pathogenic mtDNA variant will only be established with the availability of additional data.' You Might Also Like Can Apple Cider Vinegar Lead to Weight Loss? Bobbi Brown Shares Her Top Face-Transforming Makeup Tips for Women Over 50 Solve the daily Crossword

DOWNLOAD THE APP

Get Started Now: Download the App

Ready to dive into a world of global content with local flavor? Download Daily8 app today from your preferred app store and start exploring.
app-storeplay-store